Schipperheyn J J (21 results)

Language: English
Published by Springer, 2011
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
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Language: English
Published by Springer, 2011
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
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Condition: New. pp. 304.

Language: English
Published by Martinus Nijoff Publishers, 2013
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
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Paperback. Condition: Brand New. 304 pages. 9.25x6.10x0.69 inches. In Stock.

Language: English
Published by Springer Netherlands, 2011
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
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- Softcover
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- Softcover
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- Softcover
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- Softcover
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- Softcover
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Paperback. Condition: New. Softcover reprint of the original 1st ed. 1978.

Language: English
Published by Springer, 2011
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
Seller: AHA-BUCH GmbH, Einbeck, GermanyAHA-BUCH GmbH
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Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Left ventricular hypertrophy (LVH) is usually considered to be a compen satory adjustment of heart muscle to an inreased work load. LVH develops in the course of valvular or congenital heart disease, or when part of the myocardium is damaged by lon…g-standing ischemia or infarction. In the hypertrophied heart the muscle fibers increase in size, not in number. The fibers are found to contain a larger number of myofibrils and the cell organelles are larger. From epidemiologic studies it is known that even mild LVH is associated with myocardial ischemia, ventricular arrhythmias, and sudden cardiac death. Most cases of LVH show focal degenerative tissue changes including cellular atrophy, myofibrillar disorganization, interstitial fibrosis, and loss of intracellular connections. Myocardial dysfunction develops and, unlike the functional adaptive changes found in pure hypertrophy, is not reversible by surgical treatment of the valvular heart disease or medical correction of hypertension. Interstitial fibrosis, intracellular changes of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden death, a well-recognized problem in patients with a variety of heart diseases. Even When successfully treated, the patients may remain at risk if the compensatory hypertrophy is not fully reversed. Epidemiologic studies conducted in the Framingham population in the early 1950' s demonstrated LVH according to electrocardiographic criteria in 1. 5% of the population; 2% of the population had LVH according to chest X-ray criteria.

- Softcover
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- Softcover
Seller: Revaluation Books, Exeter, United KingdomRevaluation Books
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Paperback. Condition: Brand New. reprint edition. 41 pages. 9.25x6.10x0.09 inches. In Stock.

- Softcover
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Paperback. Condition: New. Softcover reprint of the original 1st ed. 1978.

Language: English
Published by Springer, 2011
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
- Print on Demand
Seller: Brook Bookstore On Demand, Napoli, NA, ItalyBrook Bookstore On Demand
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Language: English
Published by Springer, Springer Nov 2011, 2011
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
- Print on Demand
Seller: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, GermanyBuchWeltWeit Ludwig Meier e.K.
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Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Left ventricular hypertrophy (LVH) is usually considered to be a compen satory adjustment of heart muscle to an inreased work load. LVH develops in the course of valvular or congenital heart disease, or when part of the myocardium i…s damaged by long-standing ischemia or infarction. In the hypertrophied heart the muscle fibers increase in size, not in number. The fibers are found to contain a larger number of myofibrils and the cell organelles are larger. From epidemiologic studies it is known that even mild LVH is associated with myocardial ischemia, ventricular arrhythmias, and sudden cardiac death. Most cases of LVH show focal degenerative tissue changes including cellular atrophy, myofibrillar disorganization, interstitial fibrosis, and loss of intracellular connections. Myocardial dysfunction develops and, unlike the functional adaptive changes found in pure hypertrophy, is not reversible by surgical treatment of the valvular heart disease or medical correction of hypertension. Interstitial fibrosis, intracellular changes of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden death, a well-recognized problem in patients with a variety of heart diseases. Even When successfully treated, the patients may remain at risk if the compensatory hypertrophy is not fully reversed. Epidemiologic studies conducted in the Framingham population in the early 1950' s demonstrated LVH according to electrocardiographic criteria in 1. 5% of the population; 2% of the population had LVH according to chest X-ray criteria. 304 pp. Englisch.

Language: English
Published by Springer, 2011
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
- Print on Demand
Seller: Majestic Books, Hounslow, United KingdomMajestic Books
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Condition: New. Print on Demand pp. 304 Illus.

Language: English
Published by Springer, 2011
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
- Print on Demand
Seller: Biblios, frankfurt am main, HESSE, GermanyBiblios
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Condition: New. PRINT ON DEMAND pp. 304.

Language: English
Published by Springer, Springer Nov 2011, 2011
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
- Print on Demand
Seller: buchversandmimpf2000, Emtmannsberg, BAYE, Germanybuchversandmimpf2000
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Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Left ventricular hypertrophy (LVH) is usually considered to be a compen satory adjustment of heart muscle to an inreased work load. LVH develops in the course of valvular or congenital heart disease, or when part of the myocardium is da…maged by long-standing ischemia or infarction. In the hypertrophied heart the muscle fibers increase in size, not in number. The fibers are found to contain a larger number of myofibrils and the cell organelles are larger. From epidemiologic studies it is known that even mild LVH is associated with myocardial ischemia, ventricular arrhythmias, and sudden cardiac death. Most cases of LVH show focal degenerative tissue changes including cellular atrophy, myofibrillar disorganization, interstitial fibrosis, and loss of intracellular connections. Myocardial dysfunction develops and, unlike the functional adaptive changes found in pure hypertrophy, is not reversible by surgical treatment of the valvular heart disease or medical correction of hypertension. Interstitial fibrosis, intracellular changes of musc Ie cells, and loss of contract ile tissue lead to poor mechanical function and undoubtedly increase the risk of ischemia, arrhythmias, or sudden death, a well-recognized problem in patients with a variety of heart diseases. Even When successfully treated, the patients may remain at risk if the compensatory hypertrophy is not fully reversed. Epidemiologic studies conducted in the Framingham population in the early 1950' s demonstrated LVH according to electrocardiographic criteria in 1. 5% of the population; 2% of the population had LVH according to chest X-ray criteria.Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 304 pp. Englisch.
More imagesLanguage: English
Published by Springer, 2011
Series: Book 20 of 253 - Developments in Cardiovascular Medicine
- Softcover
- Print on Demand
Seller: preigu, Osnabrück, Germanypreigu
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Taschenbuch. Condition: Neu. Cardiac Left Ventricular Hypertrophy | Henk Keurs (u. a.) | Taschenbuch | Developments in Cardiovascular Medicine | 300 S. | Englisch | 2011 | Springer | EAN 9789400967618 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[…dot]com | Anbieter: preigu Print on Demand.

- Softcover
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Seller: moluna, Greven, Germanymoluna
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Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. 1 Introduction, history and contents.- 2 Isolation procedure.- 3 Perfusion box.- 4 Cardiac metabolism, electrolyte shifts and acid-base problems.- 5 Haemolysis and pulmonary edema.- 6 Electrical measurements.- 7 Haemo…dynamics.- 8 Coronary flow and oxygen co.