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Inflammatory Mechanisms in Mediating Hearing Loss
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Inflammatory Mechanisms in Mediating Hearing Loss
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Inflammatory Mechanisms in Mediating Hearing Loss
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Inflammatory Mechanisms in Mediating Hearing Loss
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Inflammatory Mechanisms in Mediating Hearing Loss
Language: English
Published by Springer Nature Switzerland AG, CH, 2019
ISBN 10: 3030064476 ISBN 13: 9783030064471
Seller: Rarewaves.com USA, London, LONDO, United Kingdom
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Paperback. Condition: New. Softcover Reprint of the Original 1st 2018 ed.
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Inflammatory Mechanisms in Mediating Hearing Loss
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Inflammatory Mechanisms in Mediating Hearing Loss
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Inflammatory Mechanisms in Mediating Hearing Loss
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Inflammatory Mechanisms in Mediating Hearing Loss
Language: English
Published by Springer International Publishing, 2019
ISBN 10: 3030064476 ISBN 13: 9783030064471
Condition: New. Details several different aspects of the cochlear inflammatory processDiscusses recent clinical trials focusing on the use of anti-inflammatory agents for the treatment of drug, noise-induced and auto-immune mediated hearing loss.
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International EditionCondition: New. Brand New! Fast Delivery This is an International Edition and ship within 24-48 hours. Deliver by FedEx and Dhl, & Aramex, UPS, & USPS and we do accept APO and PO BOX Addresses. Order can be delivered worldwide within 6-10 days and we do have flat rate for up to 2LB. Extra shipping charges will be requested if the Book weight is more than 5 LB. This Item May be shipped from India, United states & United Kingdom. Depending on your location and availability.
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Inflammatory Mechanisms in Mediating Hearing Loss
Language: English
Published by Springer International Publishing, Springer International Publishing, 2019
ISBN 10: 3030064476 ISBN 13: 9783030064471
Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Common forms of preventable hearing loss are drug and noise-induced hearing loss which are believed to be produced by a similar mechanism. The generation of reactive oxygen species appears to be a common mechanism mediating hearing loss produced by these different sources. As such, a number of laboratories have focused their research towards identifying the sources of ROS production in the cochlea following administration of chemotherapeutic agents or noise exposure. This led to the identification of ROS generating enzymes, such as xanthine oxidases, nitric oxide synthase, and NADPH oxidases which are activated and/or induced during the development of hearing loss. A consequence of these findings was the implementation of antioxidants in preclinical studies for the treatment of hearing loss. These antioxidants have provided different levels of protection in animal and human studies, but none of these have been approved by the US Food and Drug Administration for the treatmentof hearing loss. More recently, it was shown that noise-induced hearing loss was associated with recruitment of inflammatory cells and mediators in the cochlea. This finding would suggest that noise could produce injury to the cochlea which stimulates local and/or circulating inflammatory cells. A similar finding was observed in the cochlea following administration of the anticancer drug, cisplatin. In addition, our laboratory and others have provided a plausible mechanism by which noise or chemotherapeutic agents could stimulate the inflammatory response. Surprisingly, this mechanism involves ROS activation of transcription factors linked to inflammatory processes in the cochlea. These studies have led to the use of anti-inflammatory agents for the treatment of hearing loss. Preliminary studies targeting inflammatory cytokines appear especially promising in preclinical studies. A primary goal of this project is to describe our current understanding of theoxidant hypothesis of noise and drug-induced hearing loss and show how this relates to cochlear inflammation. Several different aspects of the cochlear inflammatory process will be discussed in detail, ranging from the sources of inflammatory cells, chemokines, inflammatory cytokines, and cochlea resident immune cells. Molecular pathways leading to activation of the local inflammatory process will be highlighted and treatment options will be discussed. The relevance of certain clinically used anti-inflammatory interventions, such as trans-typmanic steroids will also be discussed. Furthermore, we will examine recent patents focusing on the use of anti-inflammatory agents for the treatment of drug and noise-induced hearing loss.
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Inflammatory Mechanisms in Mediating Hearing Loss
Language: English
Published by Springer International Publishing, Springer International Publishing, 2018
ISBN 10: 3319925067 ISBN 13: 9783319925066
Buch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Common forms of preventable hearing loss are drug and noise-induced hearing loss which are believed to be produced by a similar mechanism. The generation of reactive oxygen species appears to be a common mechanism mediating hearing loss produced by these different sources. As such, a number of laboratories have focused their research towards identifying the sources of ROS production in the cochlea following administration of chemotherapeutic agents or noise exposure. This led to the identification of ROS generating enzymes, such as xanthine oxidases, nitric oxide synthase, and NADPH oxidases which are activated and/or induced during the development of hearing loss. A consequence of these findings was the implementation of antioxidants in preclinical studies for the treatment of hearing loss. These antioxidants have provided different levels of protection in animal and human studies, but none of these have been approved by the US Food and Drug Administration for the treatmentof hearing loss. More recently, it was shown that noise-induced hearing loss was associated with recruitment of inflammatory cells and mediators in the cochlea. This finding would suggest that noise could produce injury to the cochlea which stimulates local and/or circulating inflammatory cells. A similar finding was observed in the cochlea following administration of the anticancer drug, cisplatin. In addition, our laboratory and others have provided a plausible mechanism by which noise or chemotherapeutic agents could stimulate the inflammatory response. Surprisingly, this mechanism involves ROS activation of transcription factors linked to inflammatory processes in the cochlea. These studies have led to the use of anti-inflammatory agents for the treatment of hearing loss. Preliminary studies targeting inflammatory cytokines appear especially promising in preclinical studies. A primary goal of this project is to describe our current understanding of theoxidant hypothesis of noise and drug-induced hearing loss and show how this relates to cochlear inflammation. Several different aspects of the cochlear inflammatory process will be discussed in detail, ranging from the sources of inflammatory cells, chemokines, inflammatory cytokines, and cochlea resident immune cells. Molecular pathways leading to activation of the local inflammatory process will be highlighted and treatment options will be discussed. The relevance of certain clinically used anti-inflammatory interventions, such as trans-typmanic steroids will also be discussed. Furthermore, we will examine recent patents focusing on the use of anti-inflammatory agents for the treatment of drug and noise-induced hearing loss.
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Adenosine Signaling Mechanisms: Pharmacology, Functions and Therapeutic Aspects
Language: English
Published by Nova Science Pub Inc, 2015
ISBN 10: 1634831861 ISBN 13: 9781634831864
Condition: Brand New. New. US edition. Expediting shipping for all USA and Europe orders excluding PO Box. Excellent Customer Service.
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Inflammatory Mechanisms in Mediating Hearing Loss
Language: English
Published by Springer Nature Switzerland AG, CH, 2019
ISBN 10: 3030064476 ISBN 13: 9783030064471
Paperback. Condition: New. Softcover Reprint of the Original 1st 2018 ed.
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Adenosine Signaling Mechanisms: Pharmacology, Functions and Therapeutic Aspects
Language: English
Published by Nova Science Pub Inc, 2015
ISBN 10: 1634831861 ISBN 13: 9781634831864
Condition: New. This is a Brand-new US Edition. This Item may be shipped from US or any other country as we have multiple locations worldwide.
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Adenosine Signaling Mechanisms : Pharmacology, Functions and Therapeutic Aspects
Language: English
Published by Nova Science Pub Inc, 2015
ISBN 10: 1634831861 ISBN 13: 9781634831864
Seller: GreatBookPricesUK, Woodford Green, United Kingdom
Seller rating 5 out of 5 stars
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Adenosine Signaling Mechanisms: Pharmacology, Functions & Therapeutic Aspects
Language: English
Published by Nova Science Pub Inc, 2015
ISBN 10: 1634831861 ISBN 13: 9781634831864
Condition: Used. pp. 450.
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Adenosine Signaling Mechanisms : Pharmacology, Functions and Therapeutic Aspects
Language: English
Published by Nova Science Pub Inc, 2015
ISBN 10: 1634831861 ISBN 13: 9781634831864
Condition: New.
