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Biochemistry of Hypertrophy and Heart Failure (Developments in Molecular and Cellular Biochemistry, 43)
Seller: Ria Christie Collections, Uxbridge, United Kingdom
Seller rating 5 out of 5 stars
£ 96.88
£ 11.98 shipping
Ships from United Kingdom to U.S.A.Quantity: Over 20 available
Add to basketCondition: New. In.
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Condition: New.
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![Stock image for Cardiac Remodeling And Failure (Progress In Experimental Cardiology) [Hardcover] for sale by Romtrade Corp.](https://pictures.abebooks.com/isbn/9781402071775-uk-300.jpg)
Condition: New. This is a Brand-new US Edition. This Item may be shipped from US or any other country as we have multiple locations worldwide.
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Condition: Brand New. New. US edition. Expediting shipping for all USA and Europe orders excluding PO Box. Excellent Customer Service.
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Condition: As New. Unread book in perfect condition.
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Condition: New. Brand New Original US Edition. Customer service! Satisfaction Guaranteed.
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Condition: New. pp. 176.
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Paperback. Condition: Brand New. reprint edition. 172 pages. 11.00x8.27x0.42 inches. In Stock.
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Condition: New. pp. 176.
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Taschenbuch. Condition: Neu. Biochemistry of Hypertrophy and Heart Failure | Lorrie A. Kirshenbaum (u. a.) | Taschenbuch | v | Englisch | 2012 | Springer US | EAN 9781461348535 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.
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Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - The focus of this special issue of Molecular and Cellular Biochemistry is underlying mechanisms that regulate cardiac growth. The new information provided in this special issue can be utilized to design new treatment modalities that will reduce the incidence of cardiac failure which will improve quality of life in patients with chronic heart disease.
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Biochemistry of Hypertrophy and Heart Failure
Language: English
Published by Springer US, Springer US, 2003
ISBN 10: 1402074344 ISBN 13: 9781402074349
Buch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - The focus of this special issue of Molecular and Cellular Biochemistry is underlying mechanisms that regulate cardiac growth. The new information provided in this special issue can be utilized to design new treatment modalities that will reduce the incidence of cardiac failure which will improve quality of life in patients with chronic heart disease.
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Condition: New. This is a Brand-new US Edition. This Item may be shipped from US or any other country as we have multiple locations worldwide.
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Biochemistry of Apoptosis and Autophagy (Advances in Biochemistry in Health and Disease)
Book 19 of 25: Advances in Biochemistry in Health and DiseaseSeller: Ria Christie Collections, Uxbridge, United Kingdom
Seller rating 5 out of 5 stars
£ 148.08
£ 11.98 shipping
Ships from United Kingdom to U.S.A.Quantity: Over 20 available
Add to basketCondition: New. In.
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Condition: Brand New. New. US edition. Expediting shipping for all USA and Europe orders excluding PO Box. Excellent Customer Service.
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Hardcover. Condition: Like New. LIKE NEW. SHIPS FROM MULTIPLE LOCATIONS. book.
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Condition: As New. Unread book in perfect condition.
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Biology of Womens Heart Health (Hardcover)
Book 24 of 25: Advances in Biochemistry in Health and DiseaseLanguage: English
Published by Springer International Publishing AG, Cham, 2023
ISBN 10: 3031399277 ISBN 13: 9783031399275
Hardcover. Condition: new. Hardcover. Cardiovascular disease (CVD) is the leading cause of morbidity and mortality in women and men worldwide and represents a major financial burden to world health care systems. Importantly, CVD has eclipsed cancer as the leading cause of death for women globally. Through advancements in research and clinical testing, the symptoms and risk factors for CVD have been well established for men, but not for women. Consequently, there is an immediate need for new innovative research that will bridge this gap and allow for improved early diagnosis and treatment of CVD in women. This book will serve as a guide for health care providers to better understand the physiological, biochemical, and genetic differences in heart disease in women with the goal of providing improved education, awareness and treatment of cardiovascular disease in women. The book will cover topics such as: sex dependent clinical outcomes of cardiovascular disease, cardiac protection by estrogen, cardiac health during menopause, cardiac rehabilitation programs, fitness and exercise, cardio-oncology, shift work and the CVD risk, and pregnancy related CVD. Cardiovascular disease (CVD) is the leading cause of morbidity and mortality in women and men worldwide and represents a major financial burden to world health care systems. Shipping may be from multiple locations in the US or from the UK, depending on stock availability.
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Biology of Women's Heart Health (Advances in Biochemistry in Health and Disease, 26)
Book 24 of 25: Advances in Biochemistry in Health and DiseaseSeller: Ria Christie Collections, Uxbridge, United Kingdom
Seller rating 5 out of 5 stars
£ 168.56
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Biology of Women?s Heart Health
Book 24 of 25: Advances in Biochemistry in Health and DiseaseSeller: GreatBookPricesUK, Woodford Green, United Kingdom
Seller rating 5 out of 5 stars
£ 170.67
£ 15 shipping
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Taschenbuch. Condition: Neu. Biochemistry of Apoptosis and Autophagy | Lorrie A. Kirshenbaum | Taschenbuch | xvi | Englisch | 2022 | Springer | EAN 9783030788018 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.
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Condition: New. 1st ed. 2022 edition NO-PA16APR2015-KAP.
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Biochemistry Of Apoptosis And Autophagy
Book 19 of 25: Advances in Biochemistry in Health and DiseaseSeller: Kennys Bookshop and Art Galleries Ltd., Galway, GY, Ireland
Seller rating 5 out of 5 stars
Condition: New.
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Biochemistry of Apoptosis and Autophagy
Book 19 of 25: Advances in Biochemistry in Health and DiseaseLanguage: English
Published by Springer International Publishing, 2022
ISBN 10: 3030788016 ISBN 13: 9783030788018
Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - One of the most intriguing and compelling issues to impact contemporary biology to date is the concept that cell death is genetically regulated. Observations by Kerr and Wyllie, made more than 30 years ago on the basis of distinct morphological criteria, markedly distinguished apoptosis from classical cell death by necrosis. Apoptosis is a highly regulated, evolutionary conserved, genetic program of cell death essential for normal development and tissue homeostasis. The discovery of apoptosis as a regulated event and potentially amenable to therapeutic interventions has generated considerable excitement because it meant that disease entities resulting from either too much, or too little, apoptosis could be potentially cured with new therapies that target apoptosis.While there is little doubt that necrosis induced by massive cellular trauma is likely an unregulated event, several lines of investigation have challenged the dogma that necrotic cell death ismerely unregulated. Emerging data has shifted the paradigm in our thinking about necrosis as a regulated event.Autophagy is another cellular process that has received considerable attention over the past two decades and its remarkable involvement in the processes of cell survival, death and tumorigenesis. Macro autophagy is a catabolic process that involves the selective and targeted removal of oxidized proteins, macromolecular structures and organelles through an elaborate cellular process involving a lysosome mediated pathway. Other forms of autophagy involving adapter proteins, commonly referred to as chaperone mediated autophagy, involves the selective removal of cellular cargo by the ubiquitin-proteasome pathway.The book will serve as a reference guide for basic and clinical scientists who are interested in understanding how these critical cellular processes impact the pathogenesis of human disease.
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Biochemistry of Apoptosis and Autophagy
Book 19 of 25: Advances in Biochemistry in Health and DiseaseLanguage: English
Published by Springer International Publishing, 2021
ISBN 10: 3030787982 ISBN 13: 9783030787981
Buch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - One of the most intriguing and compelling issues to impact contemporary biology to date is the concept that cell death is genetically regulated. Observations by Kerr and Wyllie, made more than 30 years ago on the basis of distinct morphological criteria, markedly distinguished apoptosis from classical cell death by necrosis. Apoptosis is a highly regulated, evolutionary conserved, genetic program of cell death essential for normal development and tissue homeostasis. The discovery of apoptosis as a regulated event and potentially amenable to therapeutic interventions has generated considerable excitement because it meant that disease entities resulting from either too much, or too little, apoptosis could be potentially cured with new therapies that target apoptosis.While there is little doubt that necrosis induced by massive cellular trauma is likely an unregulated event, several lines of investigation have challenged the dogma that necrotic cell death ismerely unregulated. Emerging data has shifted the paradigm in our thinking about necrosis as a regulated event.Autophagy is another cellular process that has received considerable attention over the past two decades and its remarkable involvement in the processes of cell survival, death and tumorigenesis. Macro autophagy is a catabolic process that involves the selective and targeted removal of oxidized proteins, macromolecular structures and organelles through an elaborate cellular process involving a lysosome mediated pathway. Other forms of autophagy involving adapter proteins, commonly referred to as chaperone mediated autophagy, involves the selective removal of cellular cargo by the ubiquitin-proteasome pathway.The book will serve as a reference guide for basic and clinical scientists who are interested in understanding how these critical cellular processes impact the pathogenesis of human disease.
