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Regulation of RBP1/BCAA transcriptional repression activities
Language: English
Published by LAP LAMBERT Academic Publishing, 2013
ISBN 10: 3659350893 ISBN 13: 9783659350894
Condition: New.
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Chromatin Signaling and Diseases (Translational Epigenetics Series)
Book 5 of 39: Translational EpigeneticsLanguage: English
Published by Academic Press 2016-08-01, 2016
ISBN 10: 0128023899 ISBN 13: 9780128023891
£ 89.50
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Regulation of RBP1/BCAA transcriptional repression activities
Language: English
Published by LAP LAMBERT Academic Publishing, 2013
ISBN 10: 3659350893 ISBN 13: 9783659350894
Taschenbuch. Condition: Neu. Regulation of RBP1/BCAA transcriptional repression activities | Olivier Binda | Taschenbuch | 284 S. | Englisch | 2013 | LAP LAMBERT Academic Publishing | EAN 9783659350894 | Verantwortliche Person für die EU: BoD - Books on Demand, In de Tarpen 42, 22848 Norderstedt, info[at]bod[dot]de | Anbieter: preigu.
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Regulation Of Rbp1/Bcaa Transcriptional Repression Activities
Language: English
Published by Lap Lambert Academic Publishing, 2013
ISBN 10: 3659350893 ISBN 13: 9783659350894
Paperback. Condition: Brand New. 284 pages. 8.66x5.91x0.64 inches. In Stock.
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Chromatin Signaling and Diseases (Translational Epigenetics Series)
Book 5 of 39: Translational EpigeneticsSeller: Ria Christie Collections, Uxbridge, United Kingdom
Seller rating 5 out of 5 stars
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Chromatin Signaling and Diseases
Book 5 of 39: Translational EpigeneticsSeller: GreatBookPricesUK, Woodford Green, United Kingdom
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Hardcover. Condition: Brand New. 544 pages. 9.25x7.50x1.10 inches. In Stock.
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Paperback. Condition: Brand New. 416 pages. 9.25x7.50x0.95 inches. In Stock.
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Condition: New. 1st edition NO-PA16APR2015-KAP.
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Chromatin Signaling and Diseases
Book 5 of 39: Translational EpigeneticsSeller: GreatBookPricesUK, Woodford Green, United Kingdom
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Chromatin Readers in Health and Disease: Volume 35
Language: English
Published by Elsevier Science Publishing Co Inc, 2023
ISBN 10: 0128233761 ISBN 13: 9780128233764
Seller: THE SAINT BOOKSTORE, Southport, United Kingdom
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Chromatin Signaling and Diseases
Book 5 of 39: Translational EpigeneticsLanguage: English
Published by Elsevier Science Publishing Co Inc, US, 2016
ISBN 10: 0128023899 ISBN 13: 9780128023891
Seller: Rarewaves.com USA, London, LONDO, United Kingdom
Seller rating 5 out of 5 stars
£ 172.59
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Add to basketHardback. Condition: New. Chromatin Signaling and Diseases covers the molecular mechanisms that regulate gene expression, which govern everything from embryonic development, growth, and human pathologies associated with aging, such as cancer. This book helps researchers learn about or keep up with the quickly expanding field of chromatin signaling. After reading this book, clinicians will be more capable of explaining the mechanisms of gene expression regulation to their patients to reassure them about new drug developments that target chromatin signaling mechanisms. For example, several epigenetic drugs that act on chromatin signaling factors are in clinical trials or even approved for usage in cancer treatments, Alzheimer's, and Huntington's diseases. Other epigenetic drugs are in development to regulate various class of chromatin signaling factors. To keep up with this changing landscape, clinicians and doctors will need to stay familiar with genetic advances that translate to clinical practice, such as chromatin signaling. Although sequencing of the human genome was completed over a decade ago and its structure investigated for nearly half a century, molecular mechanisms that regulate gene expression remain largely misunderstood. An emerging concept called chromatin signaling proposes that small protein domains recognize chemical modifications on the genome scaffolding histone proteins, facilitating the nucleation of enzymatic complexes at specific loci that then open up or shut down the access to genetic information, thereby regulating gene expression. The addition and removal of chemical modifications on histones, as well as the proteins that specifically recognize these, is reviewed in Chromatin Signaling and Diseases. Finally, the impact of gene expression defects associated with malfunctioning chromatin signaling is also explored.
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Chromatin Signaling and Neurological Disorders: Volume 12
Book 18 of 39: Translational EpigeneticsLanguage: English
Published by Elsevier Science Publishing Co Inc, 2019
ISBN 10: 0128137967 ISBN 13: 9780128137963
Seller: THE SAINT BOOKSTORE, Southport, United Kingdom
Seller rating 5 out of 5 stars
£ 147.89
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Condition: New. Examines specific chromatin signaling factors that regulate spinal muscular atrophy, ulbospinal muscular atrophy, amyotrophic lateral sclerosis, Parkinson s disease, Huntington s disease, multiple sclerosis, Angelman syndrome, Rader-Willi syndrome,.
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Condition: New. Explains molecular mechanisms that regulate gene expression, which governs everything from embryonic development, growth, and human pathologies associated with aging Educates clinicians and researchers about chromatin signaling, a molecula.
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Regulation of RBP1/BCAA transcriptional repression activities
Language: English
Published by LAP LAMBERT Academic Publishing, 2013
ISBN 10: 3659350893 ISBN 13: 9783659350894
paperback. Condition: New. NEW. SHIPS FROM MULTIPLE LOCATIONS. book.
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Chromatin Signaling and Diseases
Book 5 of 39: Translational EpigeneticsLanguage: English
Published by Elsevier Science Publishing Co Inc, US, 2016
ISBN 10: 0128023899 ISBN 13: 9780128023891
£ 157.96
£ 65 shipping
Ships from United Kingdom to U.S.A.Quantity: Over 20 available
Add to basketHardback. Condition: New. Chromatin Signaling and Diseases covers the molecular mechanisms that regulate gene expression, which govern everything from embryonic development, growth, and human pathologies associated with aging, such as cancer. This book helps researchers learn about or keep up with the quickly expanding field of chromatin signaling. After reading this book, clinicians will be more capable of explaining the mechanisms of gene expression regulation to their patients to reassure them about new drug developments that target chromatin signaling mechanisms. For example, several epigenetic drugs that act on chromatin signaling factors are in clinical trials or even approved for usage in cancer treatments, Alzheimer's, and Huntington's diseases. Other epigenetic drugs are in development to regulate various class of chromatin signaling factors. To keep up with this changing landscape, clinicians and doctors will need to stay familiar with genetic advances that translate to clinical practice, such as chromatin signaling. Although sequencing of the human genome was completed over a decade ago and its structure investigated for nearly half a century, molecular mechanisms that regulate gene expression remain largely misunderstood. An emerging concept called chromatin signaling proposes that small protein domains recognize chemical modifications on the genome scaffolding histone proteins, facilitating the nucleation of enzymatic complexes at specific loci that then open up or shut down the access to genetic information, thereby regulating gene expression. The addition and removal of chemical modifications on histones, as well as the proteins that specifically recognize these, is reviewed in Chromatin Signaling and Diseases. Finally, the impact of gene expression defects associated with malfunctioning chromatin signaling is also explored.
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Chromatin Signaling and Diseases
Book 5 of 39: Translational EpigeneticsLanguage: English
Published by ACADEMIC PRESS Aug 2016, 2016
ISBN 10: 0128023899 ISBN 13: 9780128023891
Buch. Condition: Neu. Neuware.
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paperback. Condition: New. NEW. SHIPS FROM MULTIPLE LOCATIONS. book.
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Regulation of RBP1/BCAA transcriptional repression activities
Language: English
Published by LAP LAMBERT Academic Publishing Sep 2013, 2013
ISBN 10: 3659350893 ISBN 13: 9783659350894
Seller: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Germany
Seller rating 5 out of 5 stars
Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -RBP1 and BCAA repress transcription in both HDAC-independent (R1) and HDAC-dependent (R2) manners. Like RBP1, BCAA can associate with the mSIN3A/HDAC complex via the SAP30 subunit. The region responsible for this interaction (R2) mediates HDAC-dependent transcriptional repression. The latter is regulated by the NAD+-dependent enzymatic activity of the class III histone deacetylase SIRT1, which is recruited to the mSIN3A/HDAC complex via the tumour suppressors ING1b and ING2. The HDAC-independent repression activity of both RBP1 and BCAA is regulated by post-translational modifications. The R1 repression activity can be further dissected into a domain that targets basal transcription (R1 ) and one that represses both basal and activated transcription (R1s). SUMOylation of the R1s region is essential for its repression activities. SUMOylation of R1s is itself regulated by the overall local amino acids charge. The biological relevance of RBP1 and BCAA transcriptional repression activities is highlighted by their requirement for induction of cell growth arrest and terminal cell cycle withdrawal or cellular senescence. 284 pp. Englisch.
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Condition: new. Questo è un articolo print on demand.
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Hardcover. Condition: Brand New. 1st edition. 448 pages. 11.00x8.75x1.25 inches. In Stock. This item is printed on demand.
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Regulation of RBP1/BCAA transcriptional repression activities
Language: English
Published by LAP LAMBERT Academic Publishing Sep 2013, 2013
ISBN 10: 3659350893 ISBN 13: 9783659350894
Seller: buchversandmimpf2000, Emtmannsberg, BAYE, Germany
Seller rating 5 out of 5 stars
Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -RBP1 and BCAA repress transcription in both HDAC-independent (R1) and HDAC-dependent (R2) manners. Like RBP1, BCAA can associate with the mSIN3A/HDAC complex via the SAP30 subunit. The region responsible for this interaction (R2) mediates HDAC-dependent transcriptional repression. The latter is regulated by the NAD+-dependent enzymatic activity of the class III histone deacetylase SIRT1, which is recruited to the mSIN3A/HDAC complex via the tumour suppressors ING1b and ING2. The HDAC-independent repression activity of both RBP1 and BCAA is regulated by post-translational modifications. The R1 repression activity can be further dissected into a domain that targets basal transcription (R1¿) and one that represses both basal and activated transcription (R1¿). SUMOylation of the R1¿ region is essential for its repression activities. SUMOylation of R1¿ is itself regulated by the overall local amino acids charge. The biological relevance of RBP1 and BCAA transcriptional repression activities is highlighted by their requirement for induction of cell growth arrest and terminal cell cycle withdrawal or cellular senescence.VDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 284 pp. Englisch.
