Larry Hryshko (46 results)

Hardcover. Condition: Very Good. Very good condition hard cover, some wear to cover and spine, internally clean and tidy with unmarked pages, in a good readable condition.

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Condition: Brand New. New. US edition. Expediting shipping for all USA and Europe orders excluding PO Box. Excellent Customer Service.

Condition: New. This is a Brand-new US Edition. This Item may be shipped from US or any other country as we have multiple locations worldwide.

Condition: Used. pp. 204 1st Edition.

Taschenbuch. Condition: Neu. Cardiac Cell Biology | Elissavet Kardami (u. a.) | Taschenbuch | v | Englisch | 2010 | Springer US | EAN 9781441953247 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu. …

Condition: Used. pp. 204 Illus.

Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Cardiac cell biology has come of age. Recognition of activated or modified signaling molecules by specific antibodies, new selective inhibitors, and fluorescent fusion tags are but a few of the tools used to dissect signaling pathways and cross-talk mechanisms that may eventually allow rational drug design. Understanding the regulation of cardiac hypertrophy in all its complexity remains a fundamental goal of cardiac research. Since the advancement of adenovirally mediated gene transfer, transfection efficiency is no longer a limiting factor in the study of cardiomyocytes. A limiting factor in considering cell transplantion as a strategy to repair the damaged heart is cell availability at the right time. Cardiac gap junctions, intercellular communication channels that allow electrical and metabolic coupling and play an important role in arrhythmogenesis are now understood to be exquisite sensors of cardiac change. The reports in this volume incLude elegant studies that made use of cutting edge technological advances and many specialized reagents to address these issues.…

Buch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Cardiac cell biology has come of age. Recognition of activated or modified signaling molecules by specific antibodies, new selective inhibitors, and fluorescent fusion tags are but a few of the tools used to dissect signaling pathways and cross-talk mechanisms that may eventually allow rational drug design. Understanding the regulation of cardiac hypertrophy in all its complexity remains a fundamental goal of cardiac research. Since the advancement of adenovirally mediated gene transfer, transfection efficiency is no longer a limiting factor in the study of cardiomyocytes. A limiting factor in considering cell transplantion as a strategy to repair the damaged heart is cell availability at the right time. Cardiac gap junctions, intercellular communication channels that allow electrical and metabolic coupling and play an important role in arrhythmogenesis are now understood to be exquisite sensors of cardiac change. The reports in this volume incLude elegant studies that made use of cutting edge technological advances and many specialized reagents to address these issues.…

Condition: Used. pp. 204.

Condition: New. pp. 204.

Condition: New.

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Condition: As New. Unread book in perfect condition.

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Condition: New. In English.

Condition: New. In English.

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Condition: As New. Unread book in perfect condition.

Condition: New. The limitation in number of donor hearts led to the establishment of alternative treatments such as left ventricular assist devices (LVAD). This work clarifies the underlying biological mechanisms of this process, known as "reverse remodelling". Editor(s): Dhalla, Naranjan S.; Hryshko, Larry; Kardami, Elissavet; Singal, Pawan K. Series: Progress in Experimental Cardiology. Num Pages: 508 pages, biography. BIC Classification: MJD. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 234 x 156 x 28. Weight in Grams: 917. . 2003. Hardback. . . . . …

Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Cellular signaling in cardiac muscle refers to the myriad of stimuli and responses that direct and control the physiological operation of this organ. Our understand ing of these complex signaling cascades has increased dramatically over the past few decades with the advent of molecular tools for their dissection. Moreover, this infor mation is beginning to provide tangible targets towards manipulating cardiac func tion in the setting of cardiovascular disease. The mechanisms and factors that regulate cardiac cell growth are of particular interest as both adaptive and maladaptive responses can occur during cardiac hypertrophy. Cardiac hypertrophy describes the increase in individual cardiac myocyte size that is accomplished through the series and/or parallel addition of sarcomeres. The ability of cardiac muscle to increase in size through hyperplasia becomes highly restricted or negligible shortly after birth. Consequently, the increase in heart size associated with development and growth of an individual occurs through hypertrophy. In response to a chronic increase in workload, cardiac muscle cells can dramatically increase in size to face their increasing contractile demands. While this plasticity is clearly a ben eficial response under many conditions, it can be highly deleterious and inappropri ate under others. For example, cardiac hypertrophy associated with endurance exercise clearly enhances athletic performance. In contrast, the hypertrophy associated with chronic hypertension, stenotic or regurgitant heart valves, or following a myocardial infarction often continues far beyond the period where this adaptive response is ben eficial.…

Paperback. Condition: Brand New. reprint edition. 204 pages. 10.75x8.50x0.47 inches. In Stock.

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Condition: New. pp. 532.

Condition: New. The limitation in number of donor hearts led to the establishment of alternative treatments such as left ventricular assist devices (LVAD). This work clarifies the underlying biological mechanisms of this process, known as "reverse remodelling". Editor(s): Dhalla, Naranjan S.; Hryshko, Larry; Kardami, Elissavet; Singal, Pawan K. Series: Progress in Experimental Cardiology. Num Pages: 508 pages, biography. BIC Classification: MJD. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 234 x 156 x 28. Weight in Grams: 917. . 2003. Hardback. . . . . Books ship from the US and Ireland. …

Buch. Condition: Neu. Neuware - Cellular signaling in cardiac muscle refers to the myriad of stimuli and responses that direct and control the physiological operation of this organ. Our understand ing of these complex signaling cascades has increased dramatically over the past few decades with the advent of molecular tools for their dissection. Moreover, this infor mation is beginning to provide tangible targets towards manipulating cardiac func tion in the setting of cardiovascular disease. The mechanisms and factors that regulate cardiac cell growth are of particular interest as both adaptive and maladaptive responses can occur during cardiac hypertrophy. Cardiac hypertrophy describes the increase in individual cardiac myocyte size that is accomplished through the series and/or parallel addition of sarcomeres. The ability of cardiac muscle to increase in size through hyperplasia becomes highly restricted or negligible shortly after birth. Consequently, the increase in heart size associated with development and growth of an individual occurs through hypertrophy. In response to a chronic increase in workload, cardiac muscle cells can dramatically increase in size to face their increasing contractile demands. While this plasticity is clearly a ben eficial response under many conditions, it can be highly deleterious and inappropri ate under others. For example, cardiac hypertrophy associated with endurance exercise clearly enhances athletic performance. In contrast, the hypertrophy associated with chronic hypertension, stenotic or regurgitant heart valves, or following a myocardial infarction often continues far beyond the period where this adaptive response is ben eficial.…

Condition: As New. Unread book in perfect condition.