Cornelia Schuh (7 results)

Taschenbuch. Condition: Neu. Oxidative Tissue Injury in Experimental Models of Multiple Sclerosis | Cornelia Schuh | Taschenbuch | Englisch | 2015 | Südwestdeutscher Verlag für Hochschulschriften | EAN 9783838139357 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at]preigu[dot]de | Anbieter: preigu.

4°, OKart. 1. Aufl. Mit einer schönen Faltkarte in Deckeltasche, 236 S. Name auf Vorsatz, einige Anstreichungen mit Kugelschreiber, sonst schönes Ex. Sprache: Deutsch Gewicht in Gramm: 0.

Condition: Sehr gut. Band 2. 236 S.+ Kt.-Beil. (1 Bl.) [komplett] beiliegend Kopie: Abgegangene Orte im Landkreis Amberg-Sulzbach aus Hist. Verein Neumarkt Bde 29/30. 3212-3-10 Sprache: Deutsch Gewicht in Gramm: 880 4°, 27 cm x 15 cm gebundene Ausgabe, kartoniert, sehr guter Zustand, kaum Gebrauchsspuren.

Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Multiple sclerosis (MS) is a chronic disease of the central nervous system. A number of different animal models of MS contributed to a better understanding of MS pathology. Additionally, experimental models enabled the development of immune-modulatory therapies for MS patients. In recent years, growing evidence for a major role of oxidative injury in demyelination and neurodegeneration in MS has been emerging. Hence, we aimed to characterise the nature and the extent of oxidative damage in different experimental models in comparison to MS. For this purpose, we emphasised the expression of enzymes involved in reactive species production and the accumulation of oxidised phospholipids and iron, which is a potential amplifier of oxidative damage. The different animal models were triggered by diverse inflammatory mechanisms, each representing distinct aspects of MS pathology. The presented study reports a diverging extent of oxidative injury and underlying mechanisms in different models for inflammatory demyelination. 180 pp. Englisch.

Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autor/Autorin: Schuh CorneliaCornelia Schuh, PhD Postdoc at the Paracelsus Medical University in Salzburg in the Institute of Experimental and Clinical Cell Therapy PhD study at the Medical University of Vienna in the Centre for Brain Research, S.

Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Multiple sclerosis (MS) is a chronic disease of the central nervous system. A number of different animal models of MS contributed to a better understanding of MS pathology. Additionally, experimental models enabled the development of immune-modulatory therapies for MS patients. In recent years, growing evidence for a major role of oxidative injury in demyelination and neurodegeneration in MS has been emerging. Hence, we aimed to characterise the nature and the extent of oxidative damage in different experimental models in comparison to MS. For this purpose, we emphasised the expression of enzymes involved in reactive species production and the accumulation of oxidised phospholipids and iron, which is a potential amplifier of oxidative damage. The different animal models were triggered by diverse inflammatory mechanisms, each representing distinct aspects of MS pathology. The presented study reports a diverging extent of oxidative injury and underlying mechanisms in different models for inflammatory demyelination.VDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 180 pp. Englisch.

Taschenbuch. Condition: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Multiple sclerosis (MS) is a chronic disease of the central nervous system. A number of different animal models of MS contributed to a better understanding of MS pathology. Additionally, experimental models enabled the development of immune-modulatory therapies for MS patients. In recent years, growing evidence for a major role of oxidative injury in demyelination and neurodegeneration in MS has been emerging. Hence, we aimed to characterise the nature and the extent of oxidative damage in different experimental models in comparison to MS. For this purpose, we emphasised the expression of enzymes involved in reactive species production and the accumulation of oxidised phospholipids and iron, which is a potential amplifier of oxidative damage. The different animal models were triggered by diverse inflammatory mechanisms, each representing distinct aspects of MS pathology. The presented study reports a diverging extent of oxidative injury and underlying mechanisms in different models for inflammatory demyelination.