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Synopsis
Multiple sclerosis (MS) is a chronic disease of the central nervous system. A number of different animal models of MS contributed to a better understanding of MS pathology. Additionally, experimental models enabled the development of immune-modulatory therapies for MS patients. In recent years, growing evidence for a major role of oxidative injury in demyelination and neurodegeneration in MS has been emerging. Hence, we aimed to characterise the nature and the extent of oxidative damage in different experimental models in comparison to MS. For this purpose, we emphasised the expression of enzymes involved in reactive species production and the accumulation of oxidised phospholipids and iron, which is a potential amplifier of oxidative damage. The different animal models were triggered by diverse inflammatory mechanisms, each representing distinct aspects of MS pathology. The presented study reports a diverging extent of oxidative injury and underlying mechanisms in different models for inflammatory demyelination.
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Oxidative Tissue Injury in Experimental Models of Multiple Sclerosis
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Südwestdeutscher Verlag Für Hochschulschriften Feb 2015, 2015
ISBN 10: 3838139356
ISBN 13: 9783838139357
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Seller: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Germany
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Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Multiple sclerosis (MS) is a chronic disease of the central nervous system. A number of different animal models of MS contributed to a better understanding of MS pathology. Additionally, experimental models enabled the development of immune-modulatory therapies for MS patients. In recent years, growing evidence for a major role of oxidative injury in demyelination and neurodegeneration in MS has been emerging. Hence, we aimed to characterise the nature and the extent of oxidative damage in different experimental models in comparison to MS. For this purpose, we emphasised the expression of enzymes involved in reactive species production and the accumulation of oxidised phospholipids and iron, which is a potential amplifier of oxidative damage. The different animal models were triggered by diverse inflammatory mechanisms, each representing distinct aspects of MS pathology. The presented study reports a diverging extent of oxidative injury and underlying mechanisms in different models for inflammatory demyelination. 180 pp. Englisch. Seller Inventory # 9783838139357
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Oxidative Tissue Injury in Experimental Models of Multiple Sclerosis
Published by
Südwestdeutscher Verlag für Hochschulschriften, 2015
ISBN 10: 3838139356
ISBN 13: 9783838139357
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Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autor/Autorin: Schuh CorneliaCornelia Schuh, PhD Postdoc at the Paracelsus Medical University in Salzburg in the Institute of Experimental and Clinical Cell Therapy PhD study at the Medical University of Vienna in the Centre for Brain Research, S. Seller Inventory # 151605169
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Oxidative Tissue Injury in Experimental Models of Multiple Sclerosis
Published by
Südwestdeutscher Verlag Für Hochschulschriften Feb 2015, 2015
ISBN 10: 3838139356
ISBN 13: 9783838139357
New
Taschenbuch
Seller: buchversandmimpf2000, Emtmannsberg, BAYE, Germany
Seller rating 5 out of 5 stars
Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Multiple sclerosis (MS) is a chronic disease of the central nervous system. A number of different animal models of MS contributed to a better understanding of MS pathology. Additionally, experimental models enabled the development of immune-modulatory therapies for MS patients. In recent years, growing evidence for a major role of oxidative injury in demyelination and neurodegeneration in MS has been emerging. Hence, we aimed to characterise the nature and the extent of oxidative damage in different experimental models in comparison to MS. For this purpose, we emphasised the expression of enzymes involved in reactive species production and the accumulation of oxidised phospholipids and iron, which is a potential amplifier of oxidative damage. The different animal models were triggered by diverse inflammatory mechanisms, each representing distinct aspects of MS pathology. The presented study reports a diverging extent of oxidative injury and underlying mechanisms in different models for inflammatory demyelination.VDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 180 pp. Englisch. Seller Inventory # 9783838139357
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Oxidative Tissue Injury in Experimental Models of Multiple Sclerosis
Published by
Südwestdeutscher Verlag für Hochschulschriften, 2015
ISBN 10: 3838139356
ISBN 13: 9783838139357
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Seller: preigu, Osnabrück, Germany
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Taschenbuch. Condition: Neu. Oxidative Tissue Injury in Experimental Models of Multiple Sclerosis | Cornelia Schuh | Taschenbuch | Englisch | 2015 | Südwestdeutscher Verlag für Hochschulschriften | EAN 9783838139357 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at]preigu[dot]de | Anbieter: preigu. Seller Inventory # 113193987
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Oxidative Tissue Injury in Experimental Models of Multiple Sclerosis
Published by
Südwestdeutscher Verlag Für Hochschulschriften, 2015
ISBN 10: 3838139356
ISBN 13: 9783838139357
New
Taschenbuch
Seller: AHA-BUCH GmbH, Einbeck, Germany
Seller rating 5 out of 5 stars
Taschenbuch. Condition: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - Multiple sclerosis (MS) is a chronic disease of the central nervous system. A number of different animal models of MS contributed to a better understanding of MS pathology. Additionally, experimental models enabled the development of immune-modulatory therapies for MS patients. In recent years, growing evidence for a major role of oxidative injury in demyelination and neurodegeneration in MS has been emerging. Hence, we aimed to characterise the nature and the extent of oxidative damage in different experimental models in comparison to MS. For this purpose, we emphasised the expression of enzymes involved in reactive species production and the accumulation of oxidised phospholipids and iron, which is a potential amplifier of oxidative damage. The different animal models were triggered by diverse inflammatory mechanisms, each representing distinct aspects of MS pathology. The presented study reports a diverging extent of oxidative injury and underlying mechanisms in different models for inflammatory demyelination. Seller Inventory # 9783838139357