Rudolf Rott (31 results)

Condition: gut. 76 S., 21 cm, Bibliotheksexemplar. Sprache: Deutsch.

Hardcover. Condition: Very Good-. Dust Jacket Condition: No Dust Jacket. Library stamps/marks/labels/pocket, 1" tear to spine edge, otherwise light wear.; Rest of contents of Vol 85: Genetics of immunoresposiveness to natural antigens in the mouse / G. Biozzi . [et al.]; Chemistry and biology of the enterobacterial common antigen / H. Mayer, G. Schmidt; Cumulative Author and Subject Index, Volumes 40-85. ; Current Topics In Microbiology And Immunology, Vol 85; Ex-Library; Vol. 85; 184 pages.

(S. A. Hoppe-Seyler s Physiol. Chem. Bd. 363) 1982. S. 1273 - 1282. m. zahlr. Tab. br. -2) Sonderabdruck.

Condition: New. In.

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Condition: New. pp. 104.

Paperback. Condition: Brand New. 104 pages. 9.53x6.69x0.24 inches. In Stock.

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Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - The fact that none of the known DNA polymerases is able to initiate DNA chains but only to elongate from a free 3' -OH group raises the problem of how replication is initiated, both at the replication origin and on Okazaki frag ments. It was first shown by A. KORNBERG et al. that a general mechanism to initiate replication is through the formation of an RNA primer catalyzed by RNA polymerases or by a new class of enzymes, the primases (KORNBERG 1980). This mechanism, which can be used in the case of circular DNA molecules or linear DNAs that circularize or form concatemers, cannot be used at the ends of linear DNAs since the RNA primer is removed from the DNA chain, and there is no way of filling the gap resulting at the 5' -ends of the newly synthesized DNA chain. In some cases linear DNA molecules contain a palin dromic nucleotide sequence at the 3' -end that allows the formation of a hairpin structure which provides the needed free 3'-OH group for elongation. This mechanism, first proposed by CAVALIER-SMITH (1974) for eukaryotic DNA repli cation, was shown to take place in several systems (KORNBERG 1980, 1982). Another mechanism to initiate replication consists in the specific nicking of one of the strands of a circular double-stranded DNA, producing a 3'-OH group available for elongation (KORNBERG 1980).

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Condition: New. pp. ix + 185.

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Hardcover. Condition: Very Good. Very GoodDust Jacket may NOT BE INCLUDED.CDs may be missing. book.

Paperback. Condition: Brand New. reprint edition. 194 pages. 9.40x6.80x0.40 inches. In Stock.

Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Elucidation of the mechanisms of pathogenesis underlying the diseases caused by viruses and bacteria has fascinated scientists for many years in two ways. Firstly, these pathogenic agents represent relatively sim ple biological systems for the study of basic biological processes such as replication, gene regulation, genetic variability and host-pathogen interactions. Secondly, process in this field is valuable in a practi cal sence, since it can help in the control of these diseases. The avail ability of new genetic and immunological techniques, especially recom binant DNA methods and monoclonal antibody technology, has provided powerful tools for unravelling the genetic, biochemical and immunologi cal basis of viral and microbial pathogenesis. Molecular cloning has allowed the isolation of single genes or groups of genes related to phenotypes which appear to be immunologically important for pathogene sis. The specific elimination of such genes from the complex genomes of the pathogens can now be achieved with similar genetic techniques. These genetic studies have provided additional information on the role played by specific phenotypic traits in pathogenesis, especially when combined with relevant animal model systems. Furthermore, the struc tural analysis of important virulence factors and surface antigens may allow the prediction of antigenic domains suitable for the development of new vaccines. The 38th Mosbacher Colloquium focuses on the molecular basis of viral and microbial pathogenesis. The virology part begins with the well studied plant viroids. The unusual structure of their genome, as well as knowledge about their replication and pathogenicity, are presented.

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Deutsche Akademie der Naturforscher Leopoldina, Halle (Saale), 1999. 336 S., kartoniert (Einband gering fleckig)--- - Text englisch - 624 Gramm.

Condition: New. Der demografi sche Wandel ist in aller Munde. Unbestritten ist dabei,dass die Alterung unserer Gesellschaft auch fuer die Soziale Arbeit vonsteigender Bedeutung ist. Dennoch hinken Aufmerksamkeit, Interessenund Wissen der Sozialen Arbeit gegenueber dieser Ent.

Softcover. Condition: O.K. Inaugural-Dissertation zur Erlangung des Doktor-Grades bei der Veterinärmedizinischen Fakultät der Justus Liebig-Hochschule zu Gießen. 75 Seiten; Einband gebräunt/etwas bestossen/angeschmutzt, Seiten gebräunt Size: 14,5x21 cm Gewicht in Gramm: 145 Sprache: ger.

Condition: new. Questo è un articolo print on demand.

E.Postkarte in Tinte mit Ort, Datum, Unterschrift signiert R. KRZYZANOWSKI Weimar, 3.I.(19)10 (Rückseitig auf Postkarte an den Autographensammler und stud.med. (später Dr.med. / Dermatologe) Alwin Scharlau in Rostock, dem er seinen Wunsch nach seinem Autogramm erfüllt.).

Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -The fact that none of the known DNA polymerases is able to initiate DNA chains but only to elongate from a free 3' -OH group raises the problem of how replication is initiated, both at the replication origin and on Okazaki frag ments. It was first shown by A. KORNBERG et al. that a general mechanism to initiate replication is through the formation of an RNA primer catalyzed by RNA polymerases or by a new class of enzymes, the primases (KORNBERG 1980). This mechanism, which can be used in the case of circular DNA molecules or linear DNAs that circularize or form concatemers, cannot be used at the ends of linear DNAs since the RNA primer is removed from the DNA chain, and there is no way of filling the gap resulting at the 5' -ends of the newly synthesized DNA chain. In some cases linear DNA molecules contain a palin dromic nucleotide sequence at the 3' -end that allows the formation of a hairpin structure which provides the needed free 3'-OH group for elongation. This mechanism, first proposed by CAVALIER-SMITH (1974) for eukaryotic DNA repli cation, was shown to take place in several systems (KORNBERG 1980, 1982). Another mechanism to initiate replication consists in the specific nicking of one of the strands of a circular double-stranded DNA, producing a 3'-OH group available for elongation (KORNBERG 1980). 104 pp. Englisch.

Condition: New. Print on Demand pp. 104 67:B&W 6.69 x 9.61 in or 244 x 170 mm (Pinched Crown) Perfect Bound on White w/Gloss Lam.

Condition: New. PRINT ON DEMAND pp. 104.

Condition: new. Questo è un articolo print on demand.

Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -The fact that none of the known DNA polymerases is able to initiate DNA chains but only to elongate from a free 3' -OH group raises the problem of how replication is initiated, both at the replication origin and on Okazaki frag ments. It was first shown by A. KORNBERG et al. that a general mechanism to initiate replication is through the formation of an RNA primer catalyzed by RNA polymerases or by a new class of enzymes, the primases (KORNBERG 1980). This mechanism, which can be used in the case of circular DNA molecules or linear DNAs that circularize or form concatemers, cannot be used at the ends of linear DNAs since the RNA primer is removed from the DNA chain, and there is no way of filling the gap resulting at the 5' -ends of the newly synthesized DNA chain. In some cases linear DNA molecules contain a palin dromic nucleotide sequence at the 3' -end that allows the formation of a hairpin structure which provides the needed free 3'-OH group for elongation. This mechanism, first proposed by CAVALIER-SMITH (1974) for eukaryotic DNA repli cation, was shown to take place in several systems (KORNBERG 1980, 1982). Another mechanism to initiate replication consists in the specific nicking of one of the strands of a circular double-stranded DNA, producing a 3'-OH group available for elongation (KORNBERG 1980).Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 104 pp. Englisch.

Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Elucidation of the mechanisms of pathogenesis underlying the diseases caused by viruses and bacteria has fascinated scientists for many years in two ways. Firstly, these pathogenic agents represent relatively sim ple biological systems for the study of basic biological processes such as replication, gene regulation, genetic variability and host-pathogen interactions. Secondly, process in this field is valuable in a practi cal sence, since it can help in the control of these diseases. The avail ability of new genetic and immunological techniques, especially recom binant DNA methods and monoclonal antibody technology, has provided powerful tools for unravelling the genetic, biochemical and immunologi cal basis of viral and microbial pathogenesis. Molecular cloning has allowed the isolation of single genes or groups of genes related to phenotypes which appear to be immunologically important for pathogene sis. The specific elimination of such genes from the complex genomes of the pathogens can now be achieved with similar genetic techniques. These genetic studies have provided additional information on the role played by specific phenotypic traits in pathogenesis, especially when combined with relevant animal model systems. Furthermore, the struc tural analysis of important virulence factors and surface antigens may allow the prediction of antigenic domains suitable for the development of new vaccines. The 38th Mosbacher Colloquium focuses on the molecular basis of viral and microbial pathogenesis. The virology part begins with the well studied plant viroids. The unusual structure of their genome, as well as knowledge about their replication and pathogenicity, are presented. 196 pp. Englisch.

Condition: New. Print on Demand pp. ix + 185 90 Illus.

Condition: New. PRINT ON DEMAND pp. ix + 185.