Michael Elstein (23 results)

Paperback. Condition: Good. wraps are shelf rubbed and a bit chipped. light marks. small inscription. well bound. fairly good copy.[SK]. Our orders are shipped using tracked courier delivery services.

Soft cover. Condition: Very Good. The wraps are shelf rubbed.Internally clean and tightly bound.EK. Our orders are shipped using tracked courier delivery services.

Soft cover. Condition: Good. The wraps are shelf rubbed and marked.Front end page stained.No inscriptions.Well bound.EK. Our orders are shipped using tracked courier delivery services.

Hardcover. Condition: Fair. No Jacket. 512 numbered pages; minor spine crack at first free page; no dustjacket; illustrated; slightly cocked spine; light wear to corners and edges of cover; minor rub to cover; a few minor dings to cover; weighs 2 pounds and 1 ounce; measures 9.4 by 6.4 by 1.33 inches; 112624; This textbook attempts to bridge the span of pre-clinical studies, clinical finding, and management options in a readable but comprehensive manner for the medical practitioner as well as the interested non-medical reader.

Book, Paperback. Condition: Used. You Have the Power. These are USED books. Please only purchase if you are happy with that. Images & Editions We use stock images and database descriptions based on the ISBN. The image shown may NOT be the actual book cover you will receive. Covers, editions, printings, and publishers may differ. If you need a specific cover, edition, or printing, please contact us before purchasing. What ?Used? Means Because these books are pre-owned: Colouring books may be coloured in Textbooks may have answers filled in, highlighting, notes, or underlining Workbooks may have writing inside Access codes, download codes, CDs, DVDs, or online content are usually missing or already used Books that originally came with toys, inserts, maps, cards, or bonus items will usually NOT include them Pages may be folded, marked, or show normal signs of use If you are buying a book for anything other than reading (e.g. colouring, study use, collecting, gifts, specific covers, bonus items, or digital access), used books may not be suitable. Condition All books are listed as Used ? Good Books are clean and readable but not new Some books may have: Writing, highlighting, or markings Folded pages or minor wear A small warehouse or inventory sticker on the spine Original retailer stickers (we do not remove them) Our Process We purchase books by the pallet and scan thousands at a time. For this reason, we cannot photograph or individually inspect every book. Acknowledgement By purchasing, you acknowledge that: You understand the book is used You accept the condition and limitations described above Stock images and descriptions may not exactly match the item received Thank you for supporting Oak Books and helping give books a second life.

Hardcover. Condition: Fine. 9048190320 Fine/As New; Hardcover; This book is brand new and still sealed in the publisher's original shrinkwrap; Perfect, new condition; This book will be stored and delivered in a sturdy cardboard box with foam padding; Medium Format (8.5" - 9.75" tall); Dark blue and orange covers with title in white lettering; 2010, Springer-Verlag Publishing; 512 pages; "Fabry Disease," by Deborah Elstein, et al.

Condition: New. In.

Taschenbuch. Condition: Neu. Fabry Disease | Deborah Elstein (u. a.) | Taschenbuch | xxxvii | Englisch | 2014 | Springer | EAN 9789400798847 | Verantwortliche Person für die EU: Springer Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.

Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Fabry disease is an X-linked inborn error of metabolism wherein deficiency of a lysosomal enzyme results in systemic deposition of glycosphingolipids. Storage deposition, and hence pathological disease, occurs preferentially in renal glomerular and tubular epithelial cells, myocardial cells, heart valve fibrocytes, neurons of dorsal root ganglia, and in endothelial smooth muscle cells of blood vessels. Thus, Fabry disease is a multi-system disorder, albeit with considerable phenotypic heterogeneity in onset and in severity; however, it is progressive, exhibits extensive morbidity, and is life-threatening. Within the past two decades, there has been a radical change in the natural course Fabry disease by virtue of the availability of specific enzyme replacement therapy. Moreover, there has been a concerted effort to better understand the underlying pathology and equally to identify patients prior to the onset of irreversible end-organ damage. It is to be hoped that the future for patients with Fabry disease can be viewed with greater, albeit guarded, optimism. This state-of-the-art textbook attempts to bridge the span of pre-clinical studies, clinical finding, and management options in a readable but comprehensive manner for the medical practitioner as well as the interested non-medical reader.

Condition: New. pp. 512.

Condition: New. In.

Buch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - Fabry disease is an X-linked inborn error of metabolism wherein deficiency of a lysosomal enzyme results in systemic deposition of glycosphingolipids. Storage deposition, and hence pathological disease, occurs preferentially in renal glomerular and tubular epithelial cells, myocardial cells, heart valve fibrocytes, neurons of dorsal root ganglia, and in endothelial smooth muscle cells of blood vessels. Thus, Fabry disease is a multi-system disorder, albeit with considerable phenotypic heterogeneity in onset and in severity; however, it is progressive, exhibits extensive morbidity, and is life-threatening. Within the past two decades, there has been a radical change in the natural course Fabry disease by virtue of the availability of specific enzyme replacement therapy. Moreover, there has been a concerted effort to better understand the underlying pathology and equally to identify patients prior to the onset of irreversible end-organ damage. It is to be hoped that the future for patients with Fabry disease can be viewed with greater, albeit guarded, optimism. This state-of-the-art textbook attempts to bridge the span of pre-clinical studies, clinical finding, and management options in a readable but comprehensive manner for the medical practitioner as well as the interested non-medical reader.

Hardcover. Condition: Brand New. 1st edition. 512 pages. 9.25x6.25x1.25 inches. In Stock.

Condition: new. Questo è un articolo print on demand.

Condition: new. Questo è un articolo print on demand.

Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Fabry disease is an X-linked inborn error of metabolism wherein deficiency of a lysosomal enzyme results in systemic deposition of glycosphingolipids. Storage deposition, and hence pathological disease, occurs preferentially in renal glomerular and tubular epithelial cells, myocardial cells, heart valve fibrocytes, neurons of dorsal root ganglia, and in endothelial smooth muscle cells of blood vessels. Thus, Fabry disease is a multi-system disorder, albeit with considerable phenotypic heterogeneity in onset and in severity; however, it is progressive, exhibits extensive morbidity, and is life-threatening. Within the past two decades, there has been a radical change in the natural course Fabry disease by virtue of the availability of specific enzyme replacement therapy. Moreover, there has been a concerted effort to better understand the underlying pathology and equally to identify patients prior to the onset of irreversible end-organ damage. It is to be hoped that the future for patients with Fabry disease can be viewed with greater, albeit guarded, optimism. This state-of-the-art textbook attempts to bridge the span of pre-clinical studies, clinical finding, and management options in a readable but comprehensive manner for the medical practitioner as well as the interested non-medical reader. 552 pp. Englisch.

Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Fabry disease is an X-linked inborn error of metabolism wherein deficiency of a lysosomal enzyme results in systemic deposition of glycosphingolipids. Storage deposition, and hence pathological disease, occurs preferentially in renal glomerular and tubular epithelial cells, myocardial cells, heart valve fibrocytes, neurons of dorsal root ganglia, and in endothelial smooth muscle cells of blood vessels. Thus, Fabry disease is a multi-system disorder, albeit with considerable phenotypic heterogeneity in onset and in severity; however, it is progressive, exhibits extensive morbidity, and is life-threatening. Within the past two decades, there has been a radical change in the natural course Fabry disease by virtue of the availability of specific enzyme replacement therapy. Moreover, there has been a concerted effort to better understand the underlying pathology and equally to identify patients prior to the onset of irreversible end-organ damage. It is to be hoped that the future for patients with Fabry disease can be viewed with greater, albeit guarded, optimism. This state-of-the-art textbook attempts to bridge the span of pre-clinical studies, clinical finding, and management options in a readable but comprehensive manner for the medical practitioner as well as the interested non-medical reader.Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 552 pp. Englisch.

Gebunden. Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. * first full-length textbook about Fabry disease * includes chapters on the therapeutic management options * complete and stat-of-the-art range of pre-clinical studies * comprehensive chapters on clinical findings * all authors are experts with many years o.

Condition: New. Print on Demand pp. 512.

Buch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -Fabry disease is an X-linked inborn error of metabolism wherein deficiency of a lysosomal enzyme results in systemic deposition of glycosphingolipids. Storage deposition, and hence pathological disease, occurs preferentially in renal glomerular and tubular epithelial cells, myocardial cells, heart valve fibrocytes, neurons of dorsal root ganglia, and in endothelial smooth muscle cells of blood vessels. Thus, Fabry disease is a multi-system disorder, albeit with considerable phenotypic heterogeneity in onset and in severity; however, it is progressive, exhibits extensive morbidity, and is life-threatening. Within the past two decades, there has been a radical change in the natural course Fabry disease by virtue of the availability of specific enzyme replacement therapy. Moreover, there has been a concerted effort to better understand the underlying pathology and equally to identify patients prior to the onset of irreversible end-organ damage. It is to be hoped that the future for patients with Fabry disease can be viewed with greater, albeit guarded, optimism. This state-of-the-art textbook attempts to bridge the span of pre-clinical studies, clinical finding, and management options in a readable but comprehensive manner for the medical practitioner as well as the interested non-medical reader. 552 pp. Englisch.

Condition: New. PRINT ON DEMAND pp. 512.

Buch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -Fabry disease is an X-linked inborn error of metabolism wherein deficiency of a lysosomal enzyme results in systemic deposition of glycosphingolipids. Storage deposition, and hence pathological disease, occurs preferentially in renal glomerular and tubular epithelial cells, myocardial cells, heart valve fibrocytes, neurons of dorsal root ganglia, and in endothelial smooth muscle cells of blood vessels. Thus, Fabry disease is a multi-system disorder, albeit with considerable phenotypic heterogeneity in onset and in severity; however, it is progressive, exhibits extensive morbidity, and is life-threatening. Within the past two decades, there has been a radical change in the natural course Fabry disease by virtue of the availability of specific enzyme replacement therapy. Moreover, there has been a concerted effort to better understand the underlying pathology and equally to identify patients prior to the onset of irreversible end-organ damage. It is to be hoped that the future for patients with Fabry disease can be viewed with greater, albeit guarded, optimism. This state-of-the-art textbook attempts to bridge the span of pre-clinical studies, clinical finding, and management options in a readable but comprehensive manner for the medical practitioner as well as the interested non-medical reader.Springer-Verlag KG, Sachsenplatz 4-6, 1201 Wien 552 pp. Englisch.

Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. * first full-length textbook about Fabry disease * includes chapters on the therapeutic management options * complete and stat-of-the-art range of pre-clinical studies * comprehensive chapters on clinical findings * all authors are experts with many years o.