Howard B Lieberman (31 results)

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  • Language: English

    Published by Humana, 2003

    1588291154 / 9781588291158

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    hardcover. Condition: Good. Ex- library copy with stamps and stickers.

  • Language: English

    Published by Humana Press, 2003

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    Condition: Good. Pages intact with minimal writing/highlighting. The binding may be loose and creased. Dust jackets/supplements are not included. Stock photo provided. Product includes identifying sticker. Better World Books: Buy Books. Do Good.

  • Language: English

    Published by Humana, 2003

    1588291154 / 9781588291158

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    Condition: Good. Your purchase helps support Sri Lankan Children's Charity 'The Rainbow Centre'. Ex-library, so some stamps and wear, but in good overall condition. Our donations to The Rainbow Centre have helped provide an education and a safe haven to hundreds of children who live in appalling conditions.

  • Language: English

    Published by Humana, 2003

    1588291154 / 9781588291158

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    Condition: New. This is a Brand-new US Edition. This Item may be shipped from US or any other country as we have multiple locations worldwide.

  • Language: English

    Published by Humana, 2003

    1588291154 / 9781588291158

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  • Language: English

    Published by Humana, 2003

    1588291154 / 9781588291158

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  • Language: English

    Published by Humana, 2003

    1588291154 / 9781588291158

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  • Language: English

    Published by Humana Press Inc., 2010

    1617373699 / 9781617373695

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    Condition: New. Editor(s): Lieberman, Howard B. Series: Methods in Molecular Biology. Num Pages: 376 pages, biography. BIC Classification: PSF. Category: (P) Professional & Vocational. Dimension: 229 x 152 x 20. Weight in Grams: 524. . 2010. 1st ed. Softcover of orig. ed. 2004. Paperback. . . . .

  • Language: English

    Published by Humana Press Inc., 2003

    1588291154 / 9781588291158

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    Condition: New. The contributors to this text provide a detailed description of cell cycle-related methodologies using mammalian and yeast cells in the performance of experiments to further understand cell cycle checkpoint control. Editor(s): Lieberman, Howard B. Series: Methods in Molecular Biology. Num Pages: 376 pages, biography. BIC Classification: PSD. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 235 x 155 x 26. Weight in Grams: 820. . 2003. Hardback. . . . .

  • Language: English

    Published by Humana Press, 2010

    1617373699 / 9781617373695

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    Condition: New. pp. 392.

  • Language: English

    Published by Humana, 2010

    1617373699 / 9781617373695

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  • Language: English

    Published by Humana Press, 2010

    1617373699 / 9781617373695

    • Softcover

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    Paperback. Condition: Brand New. 392 pages. 9.02x5.99x0.93 inches. In Stock.

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    Language: English

    Published by Humana, 2010

    1617373699 / 9781617373695

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    Taschenbuch. Condition: Neu. Cell Cycle Checkpoint Control Protocols | Howard B. Lieberman | Taschenbuch | xvi | Englisch | 2010 | Humana | EAN 9781617373695 | Verantwortliche Person für die EU: Humana Press in Springer Science + Business Media, Heidelberger Platz 3, 14197 Berlin, juergen[dot]hartmann[at]springer[dot]com | Anbieter: preigu.

  • Language: English

    Published by Humana Pr Inc, 2003

    1588291154 / 9781588291158

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    Hardcover. Condition: Brand New. 1st edition. 372 pages. 9.00x6.00x1.00 inches. In Stock.

  • Language: English

    Published by Humana Press Inc., 2010

    1617373699 / 9781617373695

    • Softcover

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    Condition: New. Editor(s): Lieberman, Howard B. Series: Methods in Molecular Biology. Num Pages: 376 pages, biography. BIC Classification: PSF. Category: (P) Professional & Vocational. Dimension: 229 x 152 x 20. Weight in Grams: 524. . 2010. 1st ed. Softcover of orig. ed. 2004. Paperback. . . . . Books ship from the US and Ireland.

  • Language: English

    Published by Humana Press Inc., 2003

    1588291154 / 9781588291158

    • Hardcover

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    Condition: New. The contributors to this text provide a detailed description of cell cycle-related methodologies using mammalian and yeast cells in the performance of experiments to further understand cell cycle checkpoint control. Editor(s): Lieberman, Howard B. Series: Methods in Molecular Biology. Num Pages: 376 pages, biography. BIC Classification: PSD. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 235 x 155 x 26. Weight in Grams: 820. . 2003. Hardback. . . . . Books ship from the US and Ireland.

  • Language: English

    Published by Humana, 2010

    1617373699 / 9781617373695

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    Paperback. Condition: Like New. LIKE NEW. SHIPS FROM MULTIPLE LOCATIONS. book.

  • Language: English

    Published by SP SPRINGER, 2004

    1588291154 / 9781588291158

    • Hardcover
    • International Edition

    Seller: UK BOOKS STORE, London, LONDO, United KingdomUK BOOKS STORE

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    Condition: New. Brand New! Fast Delivery This is an International Edition and ship within 24-48 hours. Deliver by FedEx and Dhl, & Aramex, UPS, & USPS and we do accept APO and PO BOX Addresses. Order can be delivered worldwide within 6-10 days and we do have flat rate for up to 2LB. Extra shipping charges will be requested if the Book weight is more than 5 LB. This Item May be shipped from India, United states & United Kingdom. Depending on your location and availability.

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    Encuadernación de tapa blanda. Condition: Bien. Dust Jacket Condition: Bien. CULTURE WARS bell hooks on Spike Lee's Malcom X ECOCULTURE Andrew Ross' Weather Report GLAMOUR WOUNDS Rhonda Lieberman on The Fountainebleau Hotel FACE-LIFTS Jeanne Silverthorne on Secret Vices AMERICAN MYTHS J. Hoberman on Bill Clinton TOP TEN Greil Marcus' Real Life Rock COLUMNS FEATURES REVIEWS PROFANE ILLUMINATIONS: SOCIAL HISTORY AND THE ART OF JEFF WALL Thomas Crow WHEN IN VIDEODROME: TRAVELS IN THE NEW FLESH Howard Hampton ALEX KATZ Jack Pierson WENT LOOKING FOR AFRICA: CARRIE MAE WEEMS Kate Linker REAL TIME TRAVEL Laurence Rickels talks with Ulrike Ottinger LOCKDOWN: ROBERT GOBER AT DIA Collier Schorr THE END OF THE WORLD HAS ALREADY HAPPENED: ROBERT GOBER Donald Kuspit OPENINGS: LEONARDO DREW Hilton Als COLUMNS FEATURES REVIEWS NEW YORK Donald Kuspit on Eric Fischl Donald Kuspit on Karel Appel Donald Kuspit on A.R. Penck Keith Seward on Jenny Watson Joshua Decter on Matt Mullican Ben Lifson on Frank Majore Linda Yablonsky on Philip Pearlstein Patricia C. Phillips on Maria Nordman Patricia C. Phillips on Michelle Stuart Barry Schwabsky on Mat Collishaw Barry Schwabsky on Brenda Zlamany Jenifer P. Borum on Adolph Wölfli Jenifer P. Borum on Kenneth Goldsmith K. Marriott Jones on Stephen Ellis Ronny Cohen on Chuck Connelly Justin Spring on Sally Gall Kirby Gookin on Kirsten Mosher David Pagel on Nancy Mitchnick MILAN Francesca Pasini on Name Diffusion HARRISBURG Eileen Neff on Richard Torchia CHICAGO James Yood on Italo Scanga James Yood on Deborah Butterfield SAN FRANCISCO David Levi Strauss on Margaret Crane/Jon Winet LOS ANGELES David Pagel on Kevin Sullivan Benjamin Weissman on Thaddeus Strode Amelia Jones on José Quintana MONTREAL John K. Grande on Michèle Waquant MADRID Juan Vicente Aliaga on Francis Bacon VALENCIA Anatxu Zabalbeascoa on Joan Fontcuberta ROME Massimo Carboni on Salvatore Astore NICE Christian Kravagna on Gerwald Rockenschaub PARIS Jean-Pierre Criqui on Didier Vermeiren Olivier Zahm on Jutta Koether, Laurent Joubert ZURICH Giorgio Verzotti on Chiara Dynys, Giovanni Rizzoli VIENNA Johanna Hofleitner on Patricia London Ante Paris COLOGNE Jutta Koether on Adrian Schiess DÜSSELDORF Sabine B. Vogel on Andreas Techler HAMBURG Wolf Jahn on Klaus Kumrow LONDON Michael Corris on Gilbert & George Michael Archer on Tim Head.

  • Language: English

    Published by Humana, 2010

    1617373699 / 9781617373695

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  • Language: English

    Published by Humana Press Nov 2010, 2010

    1617373699 / 9781617373695

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    Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -The field of cell cycle regulation is based on the observation that the life cycle of a cell progresses through several distinct phases, G1, M, S, and G2, occurring in a well-defined temporal order. Details of the mechanisms involved are rapidly emerging and appear extraordinarily complex. Furthermore, not only is the order of the phases important, but in normal eukaryotic cells one phase will not begin unless the prior phase is completed successfully. Che- point control mechanisms are essentially surveillance systems that monitor the events in each phase, and assure that the cell does not progress prematurely to the next phase. If conditions are such that the cell is not ready to progress-for example, because of incomplete DNA replication in S or DNA damage that may interfere with chromosome segregation in M-a transient delay in cell cycle progression will occur. Once the inducing event is properly handled- for example, DNA replication is no longer blocked or damaged DNA is repaired-cell cycle progression continues. Checkpoint controls have recently been the focus of intense study by investigators interested in mechanisms that regulate the cell cycle. Furthermore, the relationship between checkpoint c- trol and carcinogenesis has additionally enhanced interest in these cell cycle regulatory pathways. It is clear that cancer cells often lack these checkpoints and exhibit genomic instability as a result. Moreover, several tumor suppressor genes participate in checkpoint control, and alterations in these genes are as- ciated with genomic instability as well as the development of cancer. 392 pp. Englisch.

  • Language: English

    Published by Humana Press, 2010

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    Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. The field of cell cycle regulation is based on the observation that the life cycle of a cell progresses through several distinct phases, G1, M, S, and G2, occurring in a well-defined temporal order. Details of the mechanisms involved are rapidly emerging an.

  • Language: English

    Published by Humana Press, 2003

    1588291154 / 9781588291158

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    Gebunden. Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. The field of cell cycle regulation is based on the observation that the life cycle of a cell progresses through several distinct phases, G1, M, S, and G2, occurring in a well-defined temporal order. Details of the mechanisms involved are rapidly emerging an.

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    Published by Humana Press, 2010

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    Condition: New. Print on Demand pp. 392 70 Illus. (1 Col.).

  • Language: English

    Published by Humana Press Nov 2003, 2003

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    Buch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -The field of cell cycle regulation is based on the observation that the life cycle of a cell progresses through several distinct phases, G1, M, S, and G2, occurring in a well-defined temporal order. Details of the mechanisms involved are rapidly emerging and appear extraordinarily complex. Furthermore, not only is the order of the phases important, but in normal eukaryotic cells one phase will not begin unless the prior phase is completed successfully. Che- point control mechanisms are essentially surveillance systems that monitor the events in each phase, and assure that the cell does not progress prematurely to the next phase. If conditions are such that the cell is not ready to progress¿for example, because of incomplete DNA replication in S or DNA damage that may interfere with chromosome segregation in M¿a transient delay in cell cycle progression will occur. Once the inducing event is properly handled¿ for example, DNA replication is no longer blocked or damaged DNA is repaired¿cell cycle progression continues. Checkpoint controls have recently been the focus of intense study by investigators interested in mechanisms that regulate the cell cycle. Furthermore, the relationship between checkpoint c- trol and carcinogenesis has additionally enhanced interest in these cell cycle regulatory pathways. It is clear that cancer cells often lack these checkpoints and exhibit genomic instability as a result. Moreover, several tumor suppressor genes participate in checkpoint control, and alterations in these genes are as- ciated with genomic instability as well as the development of cancer. 392 pp. Englisch.

  • Language: English

    Published by Humana Press, 2010

    1617373699 / 9781617373695

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    Condition: New. PRINT ON DEMAND pp. 392.

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    Language: English

    Published by Humana Press, 2003

    1588291154 / 9781588291158

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    Buch. Condition: Neu. Cell Cycle Checkpoint Control Protocols | Howard B. Lieberman | Buch | Einband - fest (Hardcover) | Englisch | 2003 | Humana Press | EAN 9781588291158 | Verantwortliche Person für die EU: Springer-Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg, productsafety[at]springernature[dot]com | Anbieter: preigu Print on Demand.

  • Language: English

    Published by Humana Nov 2010, 2010

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    Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -The field of cell cycle regulation is based on the observation that the life cycle of a cell progresses through several distinct phases, G1, M, S, and G2, occurring in a well-defined temporal order. Details of the mechanisms involved are rapidly emerging and appear extraordinarily complex. Furthermore, not only is the order of the phases important, but in normal eukaryotic cells one phase will not begin unless the prior phase is completed successfully. Che- point control mechanisms are essentially surveillance systems that monitor the events in each phase, and assure that the cell does not progress prematurely to the next phase. If conditions are such that the cell is not ready to progress¿for example, because of incomplete DNA replication in S or DNA damage that may interfere with chromosome segregation in M¿a transient delay in cell cycle progression will occur. Once the inducing event is properly handled¿ for example, DNA replication is no longer blocked or damaged DNA is repaired¿cell cycle progression continues. Checkpoint controls have recently been the focus of intense study by investigators interested in mechanisms that regulate the cell cycle. Furthermore, the relationship between checkpoint c- trol and carcinogenesis has additionally enhanced interest in these cell cycle regulatory pathways. It is clear that cancer cells often lack these checkpoints and exhibit genomic instability as a result. Moreover, several tumor suppressor genes participate in checkpoint control, and alterations in these genes are as- ciated with genomic instability as well as the development of cancer.Springer-Verlag GmbH, Tiergartenstr. 17, 69121 Heidelberg 392 pp. Englisch.

  • Language: English

    Published by Humana Press, Humana Press Nov 2003, 2003

    1588291154 / 9781588291158

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    Buch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -The field of cell cycle regulation is based on the observation that the life cycle of a cell progresses through several distinct phases, G1, M, S, and G2, occurring in a well-defined temporal order. Details of the mechanisms involved are rapidly emerging and appear extraordinarily complex. Furthermore, not only is the order of the phases important, but in normal eukaryotic cells one phase will not begin unless the prior phase is completed successfully. Che- point control mechanisms are essentially surveillance systems that monitor the events in each phase, and assure that the cell does not progress prematurely to the next phase. If conditions are such that the cell is not ready to progress¿for example, because of incomplete DNA replication in S or DNA damage that may interfere with chromosome segregation in M¿a transient delay in cell cycle progression will occur. Once the inducing event is properly handled¿ for example, DNA replication is no longer blocked or damaged DNA is repaired¿cell cycle progression continues. Checkpoint controls have recently been the focus of intense study by investigators interested in mechanisms that regulate the cell cycle. Furthermore, the relationship between checkpoint c- trol and carcinogenesis has additionally enhanced interest in these cell cycle regulatory pathways. It is clear that cancer cells often lack these checkpoints and exhibit genomic instability as a result. Moreover, several tumor suppressor genes participate in checkpoint control, and alterations in these genes are as- ciated with genomic instability as well as the development of cancer. 392 pp. Englisch.

  • Language: English

    Published by Humana, 2010

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    Taschenbuch. Condition: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - The field of cell cycle regulation is based on the observation that the life cycle of a cell progresses through several distinct phases, G1, M, S, and G2, occurring in a well-defined temporal order. Details of the mechanisms involved are rapidly emerging and appear extraordinarily complex. Furthermore, not only is the order of the phases important, but in normal eukaryotic cells one phase will not begin unless the prior phase is completed successfully. Che- point control mechanisms are essentially surveillance systems that monitor the events in each phase, and assure that the cell does not progress prematurely to the next phase. If conditions are such that the cell is not ready to progress-for example, because of incomplete DNA replication in S or DNA damage that may interfere with chromosome segregation in M-a transient delay in cell cycle progression will occur. Once the inducing event is properly handled- for example, DNA replication is no longer blocked or damaged DNA is repaired-cell cycle progression continues. Checkpoint controls have recently been the focus of intense study by investigators interested in mechanisms that regulate the cell cycle. Furthermore, the relationship between checkpoint c- trol and carcinogenesis has additionally enhanced interest in these cell cycle regulatory pathways. It is clear that cancer cells often lack these checkpoints and exhibit genomic instability as a result. Moreover, several tumor suppressor genes participate in checkpoint control, and alterations in these genes are as- ciated with genomic instability as well as the development of cancer.