Study Involvement Calcium Ischemic by Naz Huma (5 results)

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    • Language: English

      Published by LAP LAMBERT Academic Publishing, 2015

      3659350990 / 9783659350993

      • Softcover

      Seller: preigu, Osnabrück, Germanypreigu

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      Taschenbuch. Condition: Neu. A study on the involvement of Calcium in ischemic and normal heart | Huma Naz (u. a.) | Taschenbuch | 60 S. | Englisch | 2015 | LAP LAMBERT Academic Publishing | EAN 9783659350993 | Verantwortliche Person für die EU: preigu GmbH & Co. KG, Lengericher Landstr. 19, 49078 Osnabrück, mail[at]preigu[dot]de | Anbieter: preigu.

    • Language: English

      Published by LAP LAMBERT Academic Publishing Feb 2015, 2015

      3659350990 / 9783659350993

      • Softcover
      • Print on Demand

      Seller: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, GermanyBuchWeltWeit Ludwig Meier e.K.

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      Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -The biochemical and biophysical properties of the normal and failing human heart differ. Tension development of skinned myocardium from the failing heart has been found to be more sensitive to calcium than that from the normal heart, while maximal myofibrillar ATPase activity is depressed in the failing heart. Renal hypertension was induced in rats by employing 2-kidney-1-clip model (2K1C). Blood pressure was recorded by introducing a catheter through the left carotid artery. A Statham p23 DC transducer was used and the pressure was recorded on a Grass Polygraph. The heart was mounted and Action potential recording from papillary muscle. Myocytes were isolated from rat according to the method of Mitra and Morad (1981). The contraction of the heart were recorded for 90 min. The starting amplitude of contraction was calculated as 100% a drop in the amplitude of contraction over the 60 min was observed to be 80%. This drop in amplitude of contraction was not significant. The observations indicate that the APD 40 is most sensititve to calcium alteration in the superfused buffer. The rat myocytes were studied by flow-cytometr. 60 pp. Englisch.

    • Language: English

      Published by LAP LAMBERT Academic Publishing, 2015

      3659350990 / 9783659350993

      • Softcover
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      Seller: moluna, Greven, Germanymoluna

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      Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Autor/Autorin: Naz HumaDr. Huma Naz, Aligarh Muslim University Aligarh, U.P., India.Ph.D. 2013: Aligarh Muslim University (AMU), Aligarh, U.P., India. M.Sc. (Microbiology)2005 : Hemwati Nandan Bhuguna, Garhwal University, Garhwal, India.PUBLICATIO.

    • Language: English

      Published by LAP LAMBERT Academic Publishing, 2015

      3659350990 / 9783659350993

      • Softcover
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      Seller: AHA-BUCH GmbH, Einbeck, GermanyAHA-BUCH GmbH

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      Taschenbuch. Condition: Neu. nach der Bestellung gedruckt Neuware - Printed after ordering - The biochemical and biophysical properties of the normal and failing human heart differ. Tension development of skinned myocardium from the failing heart has been found to be more sensitive to calcium than that from the normal heart, while maximal myofibrillar ATPase activity is depressed in the failing heart. Renal hypertension was induced in rats by employing 2-kidney-1-clip model (2K1C). Blood pressure was recorded by introducing a catheter through the left carotid artery. A Statham p23 DC transducer was used and the pressure was recorded on a Grass Polygraph. The heart was mounted and Action potential recording from papillary muscle. Myocytes were isolated from rat according to the method of Mitra and Morad (1981). The contraction of the heart were recorded for 90 min. The starting amplitude of contraction was calculated as 100% a drop in the amplitude of contraction over the 60 min was observed to be 80%. This drop in amplitude of contraction was not significant. The observations indicate that the APD 40 is most sensititve to calcium alteration in the superfused buffer. The rat myocytes were studied by flow-cytometr.

    • Language: English

      Published by LAP LAMBERT Academic Publishing Feb 2015, 2015

      3659350990 / 9783659350993

      • Softcover
      • Print on Demand

      Seller: buchversandmimpf2000, Emtmannsberg, BAYE, Germanybuchversandmimpf2000

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      Taschenbuch. Condition: Neu. This item is printed on demand - Print on Demand Titel. Neuware -The biochemical and biophysical properties of the normal and failing human heart differ. Tension development of skinned myocardium from the failing heart has been found to be more sensitive to calcium than that from the normal heart, while maximal myofibrillar ATPase activity is depressed in the failing heart. Renal hypertension was induced in rats by employing 2-kidney-1-clip model (2K1C). Blood pressure was recorded by introducing a catheter through the left carotid artery. A Statham p23 DC transducer was used and the pressure was recorded on a Grass Polygraph. The heart was mounted and Action potential recording from papillary muscle. Myocytes were isolated from rat according to the method of Mitra and Morad (1981). The contraction of the heart were recorded for 90 min. The starting amplitude of contraction was calculated as 100% a drop in the amplitude of contraction over the 60 min was observed to be 80%. This drop in amplitude of contraction was not significant. The observations indicate that the APD 40 is most sensititve to calcium alteration in the superfused buffer. The rat myocytes were studied by flow-cytometr.VDM Verlag, Dudweiler Landstraße 99, 66123 Saarbrücken 60 pp. Englisch.