Novartis Foundation Symposia: Cancer and Inflammation
Language: English
Published by WILEY, 2004
- Hardcover
- Used
Condition: Used - Fine
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Leichte Gebrauchsspuren; Leichte Verschmutzung / Farbtonveränderung. Chronic inflammation is linked to certain cancers, with the host's response to malignancy sharing similarities with inflammation and wound healing. Inflammation-related cells, cytokines, and chemokine systems are present in various common cancers. Neutralizing or deleting genes for specific inflammatory cytokines can provide resistance to tumor formation and metastasis, while over-expressing these cytokines in tumor cells may increase malignancy. Some chemokines may hinder antitumor immunity by promoting ineffective Type 2 responses, and tumor cells can exploit chemokine receptors to migrate to lymph nodes and other organs. Consequently, the components associated with inflammation in tumors tend to facilitate tumor growth, progression, and immunosuppression rather than an effective antitumor response. This book compiles insights from an international array of scientists and clinicians across fields such as epidemiology, immunology, cell biology, molecular oncology, and pharmacology to explore these topics. Discussions include the epidemiological connections between cancer and inflammation, the role of inflammation in cancer development, inflammatory genes as cancer risk factors, the relationship between inflammation and cancer angiogenesis, and strategies for prevention and treatment.…
Seller Inventory # 8d84bd59-253c-45e2-b436-c61f577fc2af
- Title
- Novartis Foundation Symposia: Cancer and Inflammation
- Author
- Novartis Foundation
- Publisher
- WILEY
- Publication year
- 2004
- Condition
- Fine
- Binding
- Hardcover
- Language
- English
- ISBN 10
- 047085510X
- ISBN 13
- 9780470855102
Neutralization or deletion of the gene for some inflammatory cytokines confers resistance to tumour induction and experimental metastasis. Over-expression of such cytokines in tumour cells may enhance malignant potential. Certain chemokines are likely to subvert antitumour immunity by favouring development of ineffective Type 2 responses. Tumour cells may even utilize chemokine receptors in homing to lymph nodes and other organs. Thus, the cells, cytokines and chemokines found in tumours are more likely to contribute to tumour growth, progression and immunosuppression than they are to mount an effective host antitumour response.
This book draws together contributions from an international group of scientists and clinicians from diverse disciplines, ranging from epidemiology to immunology, cell biology, molecular oncology, molecular medicine and pharmacology to debate these and related issues. Topics covered include the epidemiological links between cancer and inflammation, the parallels between inflammation and cancer, the role of inflammation in cancer, inflammatory genes as risk factors for cancer initiation and progression, inflammation and cancer angiogenesis, and preventative and therapeutic strategies.
Related Novartis Foundation symposia:
252 Generation and Effector Functions of Regulatory Lymphocytes
Chair: Jean-François Bach
254 Immunoinformatics: Bioinformatic Strategies for Better Understanding of Immune Function
Chair: Hans-Georg Rammensee
"Synopsis" may belong to another edition of this title.
About the Author
Chair: SIAMON GORDON, Sir William Dunn School of Pathology, University of Oxford, UK
"About the title" may belong to another edition of this title.
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