Model Systems in Aging
Language: English
Published by Springer Berlin Heidelberg, 2003
- Hardcover
- New
Condition: New
£ 154.73
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Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. The unique feature of this book is its comparative approach to different genetic model systemsFull text available online for subscribers of the seriesAging is the progressive decline in biological functions over time. This decline targe.
Seller Inventory # 4877864
- Title
- Model Systems in Aging
- Author
- Nyström, T.|Osiewacz, H. D.
- Publisher
- Springer Berlin Heidelberg
- Publication year
- 2003
- Condition
- New
- Binding
- Gebunden
- Language
- English
- ISBN 10
- 3540024905
- ISBN 13
- 9783540024903
- Series
- Book 1 of 17: Topics in Current Genetics
- Seller catalogs
- Mathematik/Naturwissenschaften/Technik/Medizin
Aging is the progressive decline in biological functions over time. This decline targets macromolecules, cells, tissues and, as a consequence, whole organisms. Despite considerable progress in the development of testable hypothesis concerning aging in an evolutionary context, a unifying theory of the molecular/physiological mechanistic causes of aging has not been reached. In fact, is it not clear to what extent aging is a programmed or stochastic process.
This book takes the reader from unicellular bacterial deterioration via senescence in fungi and worms to aging in rodents and humans, allowing a comparative view on similarities and differences in different genetic model systems. The different model systems are scrutinized in the light of contemporary aging hypothesis, such as the free radical and genomic instability theories.
"Synopsis" may belong to another edition of this title.
From the Back Cover
Aging is the progressive decline in biological functions over time. This decline targets macromolecules, cells, tissues and, as a consequence, whole organisms. Despite considerable progress in the development of testable hypothesis concerning aging in an evolutionary context, a unifying theory of the molecular/physiological mechanistic causes of aging has not been reached. In fact, is it not clear to what extent aging is a programmed or stochastic process.
This book takes the reader from unicellular bacterial deterioration via senescence in yeast and worms to aging in rodents and humans, allowing a comparative view on similarities and differences in different genetic model systems. The different model systems are scrutinized in the light of contemporary aging hypothesis, such as the free radical and genomic instability theories.
"About the title" may belong to another edition of this title.
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