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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: Basi6 International, Irving, TX, U.S.A.
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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: Anis Press, Walnut Creek, CA, U.S.A.
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Hardcover Jan 14, 2009. Condition: New.
Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: Books Puddle, New York, NY, U.S.A.
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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: Majestic Books, Hounslow, United Kingdom
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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: GreatBookPrices, Columbia, MD, U.S.A.
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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: booksXpress, Bayonne, NJ, U.S.A.
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Published by Springer, 2010
ISBN 10: 9048181275ISBN 13: 9789048181278
Seller: booksXpress, Bayonne, NJ, U.S.A.
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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: Ria Christie Collections, Uxbridge, United Kingdom
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Published by Springer, 2010
ISBN 10: 9048181275ISBN 13: 9789048181278
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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
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Published by Springer, 2010
ISBN 10: 9048181275ISBN 13: 9789048181278
Seller: Lucky's Textbooks, Dallas, TX, U.S.A.
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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
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Published by Springer Netherlands Jan 2009, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Germany
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Buch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably. 296 pp. Englisch.
Published by Springer Netherlands Okt 2010, 2010
ISBN 10: 9048181275ISBN 13: 9789048181278
Seller: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Germany
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Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably. 292 pp. Englisch.
Published by Springer Netherlands, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: moluna, Greven, Germany
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Gebunden. Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Will reflect the recent advances and exponential growth of the field of neurodegenerationWill also provoke questions related to the molecular base and diversity of initiation of conformational diseases up to their pathological classification.
Published by Springer Netherlands, 2010
ISBN 10: 9048181275ISBN 13: 9789048181278
Seller: moluna, Greven, Germany
Book Print on Demand
Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. Will reflect the recent advances and exponential growth of the field of neurodegenerationWill also provoke questions related to the molecular base and diversity of initiation of conformational diseases up to their pathological classification.
Published by Springer Netherlands, 2010
ISBN 10: 9048181275ISBN 13: 9789048181278
Seller: AHA-BUCH GmbH, Einbeck, Germany
Book
Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably.
Published by Springer Netherlands, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: AHA-BUCH GmbH, Einbeck, Germany
Book
Buch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - It was twenty ve years ago this year that for the rst time a protein under- ing a form of human cerebral amyloidosis, the Icelandic-type hereditary cerebral haemorrhage was identi ed. This, together with the recognition that an amino acid substitution can transform the wild type cystatin C into a disease-associated amyloid-forming protein in this condition, was only a prelude to a series of imp- tant discoveries that followed. As a result, pathologically altered proteins have been brought into the centre stage of research into the pathomechanism of a n- ber of neurodegenerative diseases, which include epidemiologically such important conditions as Alzheimer s disease or Parkinson s disease and, among others, also the transmissible spongiform encephalopathies, Huntington s chorea, spinocereb- lar ataxias, frontotemporal lobar degenerations and amyotrophic lateral sclerosis. Despite the diversity in the amino acid sequence of the different proteins involved in these neurological diseases, one of the common themes underlying the patho- chanisms of all these conditions is protein misfolding, aggregation hence the term protein folding disorders , which can trigger cascades of events ultimately resulting in synapse loss and neuron death with devastating clinical consequences in many of the most precious spheres of human existence including personality, cognition, memory, skilled movements and affection. It is always a challenging task to unite the different topics of the individual ch- ters into a common theme in a multi-author volume, but the current book edited by Judit Ovadi and Ferenc Orosz ts this task admirably.
Published by Springer, 2010
ISBN 10: 9048181275ISBN 13: 9789048181278
Seller: Revaluation Books, Exeter, United Kingdom
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Paperback. Condition: Brand New. 288 pages. 9.25x6.10x0.66 inches. In Stock.
Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: GreatBookPricesUK, Castle Donington, DERBY, United Kingdom
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Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: Mispah books, Redhill, SURRE, United Kingdom
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Hardcover. Condition: Like New. Like New. book.
Published by Springer, 2009
ISBN 10: 1402094337ISBN 13: 9781402094330
Seller: GreatBookPrices, Columbia, MD, U.S.A.
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Condition: As New. Unread book in perfect condition.