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Published by Springer, 2012
ISBN 10: 3642953107ISBN 13: 9783642953101
Seller: International Book Project, Lexington, KY, U.S.A.
Book
Paperback. Condition: Very Good. Book is in very good condition. Signs of wear on the outside dust cover edges from storage, but everything is clean and bright. No writing and highlighting in the interior. Great copy! Ships quickly! 100% of the book sales go towards furthering the International Book Project's mission of promoting literacy in the developing world.
Published by Springer, 2012
ISBN 10: 3642953107ISBN 13: 9783642953101
Seller: booksXpress, Bayonne, NJ, U.S.A.
Book Print on Demand
Soft Cover. Condition: new. This item is printed on demand.
Published by Springer, 2012
ISBN 10: 3642953107ISBN 13: 9783642953101
Seller: Lucky's Textbooks, Dallas, TX, U.S.A.
Book
Condition: New.
Published by Springer, 2012
ISBN 10: 3642953107ISBN 13: 9783642953101
Seller: Ria Christie Collections, Uxbridge, United Kingdom
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Condition: New. PRINT ON DEMAND Book; New; Fast Shipping from the UK. No. book.
Published by Springer Verlag, 2012
ISBN 10: 3642953107ISBN 13: 9783642953101
Seller: Revaluation Books, Exeter, United Kingdom
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Paperback. Condition: Brand New. 852 pages. 9.61x6.69x1.69 inches. In Stock.
Published by Springer Berlin Heidelberg, 2012
ISBN 10: 3642953107ISBN 13: 9783642953101
Seller: AHA-BUCH GmbH, Einbeck, Germany
Book
Taschenbuch. Condition: Neu. Druck auf Anfrage Neuware - Printed after ordering - The presence of monotypism in thick atherosclerotic lesions of black females with G-6-PD mosaicism first reported by the Benditts (1973) has been confirmed in two other laboratories. However, we believe that it is premature to conclude that the finding of monotypism necessarily indicates monoclonal origin of athero sclerotic lesions. We have suggested two alternative explanations for the obser vation of monotypism which we believe must be shown to be invalid before accept ing monoclonal origin as the only plausible way to account for the observed G-6-PD monotypism. One of these two alternatives relates to clonal heterogeneity of cell growth potential, i. e. , during the course of progressive growth of a le sion, progeny of one cell may overgrow all others in a portion of the lesion. The other alternative is that one of the G-6-PD alleles may be linked to genes that afford a preferential survival characteristic in the abnormal environment present in atheroscerotic lesions. Thus, cells with one allele may be able to grow better than cells with the other allele, and this characteristic may be unrelated to 'A-ness' or 'B-ness'. We have studied initiation of lesions in He diet-fed swine and demonstrated that all active lesions that were studied were of multiple cell origin (not monoclo nal). We have studied cell growth patterns in developing atherosclerotic lesions in He diet-fed swine and found evidence consistent with clonal heterogeneity in growth potential of lesion cells.
Published by Springer Berlin Heidelberg, 2012
ISBN 10: 3642953107ISBN 13: 9783642953101
Seller: moluna, Greven, Germany
Book Print on Demand
Condition: New. Dieser Artikel ist ein Print on Demand Artikel und wird nach Ihrer Bestellung fuer Sie gedruckt. The presence of monotypism in thick atherosclerotic lesions of black females with G-6-PD mosaicism first reported by the Benditts (1973) has been confirmed in two other laboratories. However, we believe that it is premature to conclude that the finding of m.
Published by Springer 2012-02, 2012
ISBN 10: 3642953107ISBN 13: 9783642953101
Seller: Chiron Media, Wallingford, United Kingdom
Book
PF. Condition: New.
Published by Springer Berlin Heidelberg Feb 2012, 2012
ISBN 10: 3642953107ISBN 13: 9783642953101
Seller: BuchWeltWeit Ludwig Meier e.K., Bergisch Gladbach, Germany
Book Print on Demand
Taschenbuch. Condition: Neu. This item is printed on demand - it takes 3-4 days longer - Neuware -The presence of monotypism in thick atherosclerotic lesions of black females with G-6-PD mosaicism first reported by the Benditts (1973) has been confirmed in two other laboratories. However, we believe that it is premature to conclude that the finding of monotypism necessarily indicates monoclonal origin of athero sclerotic lesions. We have suggested two alternative explanations for the obser vation of monotypism which we believe must be shown to be invalid before accept ing monoclonal origin as the only plausible way to account for the observed G-6-PD monotypism. One of these two alternatives relates to clonal heterogeneity of cell growth potential, i. e. , during the course of progressive growth of a le sion, progeny of one cell may overgrow all others in a portion of the lesion. The other alternative is that one of the G-6-PD alleles may be linked to genes that afford a preferential survival characteristic in the abnormal environment present in atheroscerotic lesions. Thus, cells with one allele may be able to grow better than cells with the other allele, and this characteristic may be unrelated to 'A-ness' or 'B-ness'. We have studied initiation of lesions in He diet-fed swine and demonstrated that all active lesions that were studied were of multiple cell origin (not monoclo nal). We have studied cell growth patterns in developing atherosclerotic lesions in He diet-fed swine and found evidence consistent with clonal heterogeneity in growth potential of lesion cells. 852 pp. Englisch.
Published by Springer, 2012
ISBN 10: 3642953107ISBN 13: 9783642953101
Seller: Mispah books, Redhill, SURRE, United Kingdom
Book
Paperback. Condition: Like New. Like New. book.