In Neuroglia in Alzheimer's Disease, the authors provide a broad overview of glial physiology and pathophysiology, outline glial contribution to neurodegeneration and concentrate on the role of different classes of neuroglial cells in the progression and outcome of Alzheimer's disease. The authors discuss the critical vie of AD and argue for glial cells as new targets for therapy of neurodegenerative disorders. This book is valuable researchers, clinicians, postdocs, and graduate students in the fields of neurobiology, neurology and psychiatry. . The first comprehensive overview of the contribution of various types of glia into Alzheimer's Disease. . Provides a new concept of gliopathology that can influence understanding of the disease and the development of new therapeutic strategies . Written by an international team of leading researchers in the field of neuroglia
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Dr. Verkhratsky is a Professor of Neurophysiology, Faculty of Life Sciences at the University of Manchester. He is currently serving as Editor-in-Chief for Cell Calcium and Membrane Transport & Signalling and is on the Editorial Board of over 12 journals. He was previously a Chairman for the Division of Neuroscience at University of Manchester. He has published over 280 journal articles. Vladimir Parpura, M.D., Ph.D holds both a medical degree, awarded from the University of Zagreb in Croatia in 1989, and a doctorate, received in Neuroscience and Zoology from Iowa State University in 1993. He has held faculty appointments at the Department of Zoology and Genetics, Iowa State University and the Department of Cell Biology and Neuroscience, University of California Riverside. He is presently an Associate Professor in the Department of Neurobiology, University of Alabama Birmingham. Dr. Arellano is a Research Professor at the University of Basque Country in Bizkaia, Spain. He founded the Functional Neuroanatomy Laboratory in 2010 at University of Basque Country. His research focuses on Researching on the neuroanatomy and functional connectivity of neural circuits in the context of memory-related plasticity under normal and pathological conditions.
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