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International Review of Neurobiology: 42: Volume 42 - Hardcover

 
9780123668424: International Review of Neurobiology: 42: Volume 42
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Volume 42 presents an in-depth review on Alzheimer's Disease as well as a look at several transcription factors.

  • Clinical features of stroke and the current stasis of stroke therapy
  • Alzheimers Disease--pathology, pathological proteins, etiology, pathogenesis, neurotransmitter alterations, and treatment strategies
  • An examination of the regulation of AP-1 and related transcription factors and their role in brain injury and drug abuse-related behavior
  • Emerging roles of the transcription factor NF(B
  • Recent advances about the nature of alterations in ion channel structure or function which may be responsible for epileptogenesis
  • An integrated model accounting for the multiple mechanisms involved in the posttranslational regulation of glutamate ionotroic receptors
  • Genetic mutation and the Glycine Receptor

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Review:
International Review of Neurobiology is a well-respected series appealing to neuroscientists, clinicians, psychologists, physiologists, and pharmacologists. Led by an internationally renowned editorial board, this important serial publishes both eclectic volumes made up of timely reviews and thematic volumes that focus on recent progress in a specific area of neurobiology research. This volume is a cumulative subject index of volumes 26-50.
Synopsis:
Volume 42 presents an in-depth review on Alzheimer's Disease as well as a look at several transcription factors. Among its key features, it: provides clinical features of stroke and the current stasis of stroke therapy; talks about Alzheimer's Disease - pathology, pathological proteins, etiology, pathogenesis, neurotransmitter alterations, and treatment strategies; presents an examination of the regulation of AP-1 and related transcription factors and their role in brain injury and drug abuse-related behavior; talks about emerging roles of the transcription factor NF (B Recent advances about the nature of alterations in ion channel structure or function which may be responsible for epileptogenesis; presents an integrated model accounting for the multiple mechanisms involved in the posttranslational regulation of glutamate ionotroic receptors; and covers Genetic mutation and the Glycine Receptor.

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